Abnormalities of calcium metabolism and myocardial contractility depression in the failing heart.
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Diabetic cardiomyopathy: signaling defects and therapeutic approachesImpaired Ca(2+)-handling in HIF-1alpha(+/-) mice as a consequence of pressure overload.Early remodeling of perinuclear Ca2+ stores and nucleoplasmic Ca2+ signaling during the development of hypertrophy and heart failure.{beta}1-Adrenergic receptor activation induces mouse cardiac myocyte death through both L-type calcium channel-dependent and -independent pathways.Selective inhibitors of cardiac ADPR cyclase as novel anti-arrhythmic compounds.Increased cardiac myocyte PDE5 levels in human and murine pressure overload hypertrophy contribute to adverse LV remodelingNuclear calcium in cardiac myocytes.Critical role for stromal interaction molecule 1 in cardiac hypertrophyIn search of new therapeutic targets and strategies for heart failure: recent advances in basic scienceA new approach to assessment of the left ventricleDivide and conquer: the application of organelle proteomics to heart failure.Left ventricular remodeling in the post-infarction heart: a review of cellular, molecular mechanisms, and therapeutic modalities.Use of inotropic agents in patients with advanced heart failure: lessons from recent trials and hopes for new agents.Ca(2+) signaling: an outlook on the characterization of Ca(2+) channels and their importance in cellular functionsThe role of 3D wall motion tracking in heart failure.Mechanisms of disease: is mitochondrial function altered in heart failure?SERCA2a: a prime target for modulation of cardiac contractility during heart failure.Characterization of Ca(2+)-Dependent Protein-Protein Interactions within the Ca(2+) Release Units of Cardiac Sarcoplasmic Reticulum.A Review of the Molecular Mechanisms Underlying the Development and Progression of Cardiac Remodeling.Raf kinase inhibitor protein: lessons of a better way for β-adrenergic receptor activation in the heart.Metformin prevents the development of chronic heart failure in the SHHF rat model.Coupling of the Functional Stability of Rat Myocardium and Activity of Lipid Peroxidation in Combined Development of Postinfarction Remodeling and Diabetes MellitusModulation of SR Ca2+ release by the triadin-to-calsequestrin ratio in ventricular myocytes.Mitochondrial uncoupling downregulates calsequestrin expression and reduces SR Ca2+ stores in cardiomyocytes.Cardiac RKIP induces a beneficial β-adrenoceptor-dependent positive inotropy.Beneficial effects of leptin treatment in a setting of cardiac dysfunction induced by transverse aortic constriction in mouse.Pivotal role of α2 Na+ pumps and their high affinity ouabain binding site in cardiovascular health and disease.Urocortin-2 Prevents Dysregulation of Ca2+ Homeostasis and Improves Early Cardiac Remodeling After Ischemia and Reperfusion.Calcitriol, the Bioactive Metabolite of Vitamin D, Increases Ventricular K Currents in Isolated Mouse CardiomyocytesInfluence of Amiodarone and Dronedarone on the Force-Interval Dependence of Rat Myocardium
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P2860
Abnormalities of calcium metabolism and myocardial contractility depression in the failing heart.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on December 2009
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vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Abnormalities of calcium metab ...... pression in the failing heart.
@en
Abnormalities of calcium metab ...... pression in the failing heart.
@nl
type
label
Abnormalities of calcium metab ...... pression in the failing heart.
@en
Abnormalities of calcium metab ...... pression in the failing heart.
@nl
prefLabel
Abnormalities of calcium metab ...... pression in the failing heart.
@en
Abnormalities of calcium metab ...... pression in the failing heart.
@nl
P2860
P1476
Abnormalities of calcium metab ...... pression in the failing heart.
@en
P2093
Lars S Maier
Stephan E Lehnart
P2860
P2888
P304
P356
10.1007/S10741-009-9146-X
P577
2009-12-01T00:00:00Z