Cardiac Specific Overexpression of Mitochondrial Omi/HtrA2 Induces Myocardial Apoptosis and Cardiac Dysfunction. - Wikidata - wikimedia - TriplyDB

Cardiac Specific Overexpression of Mitochondrial Omi/HtrA2 Induces Myocardial Apoptosis and Cardiac Dysfunction.

Cardiac Specific Overexpression of Mitochondrial Omi/HtrA2 Induces Myocardial Apoptosis and Cardiac Dysfunction.

http://www.wikidata.org/entity/Q37479622

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Expression of p53 in myocardium following coronary microembolization in rats and its significance.Melatonin and mitochondrial function during ischemia/reperfusion injury.The mitochondria-targeting peptide elamipretide diminishes circulating HtrA2 in ST-segment elevation myocardial infarction.

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Cardiac Specific Overexpression of Mitochondrial Omi/HtrA2 Induces Myocardial Apoptosis and Cardiac Dysfunction.

http://www.wikidata.org/entity/Q37479622

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on 07 December 2016

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Cardiac Specific Overexpressio ...... tosis and Cardiac Dysfunction.

@en

Cardiac Specific Overexpressio ...... tosis and Cardiac Dysfunction.

@nl

type

label

Cardiac Specific Overexpressio ...... tosis and Cardiac Dysfunction.

@en

Cardiac Specific Overexpressio ...... tosis and Cardiac Dysfunction.

@nl

prefLabel

Cardiac Specific Overexpressio ...... tosis and Cardiac Dysfunction.

@en

Cardiac Specific Overexpressio ...... tosis and Cardiac Dysfunction.

@nl

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Scientific Reports

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Cardiac Specific Overexpressio ...... tosis and Cardiac Dysfunction.

@en

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Huirong Liu

http://www.w3.org/2001/XMLSchema#string

Jianyu Shang

http://www.w3.org/2001/XMLSchema#string

Ke Wang

http://www.w3.org/2001/XMLSchema#string

Kun Jiao

http://www.w3.org/2001/XMLSchema#string

Lin Zuo

http://www.w3.org/2001/XMLSchema#string

Lu Ma

http://www.w3.org/2001/XMLSchema#string

Wayne Bond Lau

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Wen Wang

http://www.w3.org/2001/XMLSchema#string

Xiaoliang Wang

http://www.w3.org/2001/XMLSchema#string

Xin Liu

http://www.w3.org/2001/XMLSchema#string

P2860

Proteome-wide Identification of HtrA2/Omi Substrates

Effects of aging and exercise training on apoptosis in the heart

Mitochondrial protease Omi/HtrA2 enhances caspase activation through multiple pathways

Osteopontin: At the cross-roads of myocyte survival and myocardial function

The mitochondrial serine protease HtrA2/Omi: an overview

Inhibition of HtrA2/Omi ameliorates heart dysfunction following ischemia/reperfusion injury in rat heart in vivo

The serine protease Omi/HtrA2 is involved in XIAP cleavage and in neuronal cell death following focal cerebral ischemia/reperfusion

Variations in the protein level of Omi/HtrA2 in the heart of aged rats may contribute to the increased susceptibility of cardiomyocytes to ischemia/reperfusion injury and cell death : Omi/HtrA2 and aged heart injury

p53-mediated activation of the mitochondrial protease HtrA2/Omi prevents cell invasion.

Overexpression of HAX-1 protects cardiac myocytes from apoptosis through caspase-9 inhibition.

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37927

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scholarly article

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10.1038/SREP37927

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6

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2016-12-07T00:00:00Z

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