Variations in the protein level of Omi/HtrA2 in the heart of aged rats may contribute to the increased susceptibility of cardiomyocytes to ischemia/reperfusion injury and cell death : Omi/HtrA2 and aged heart injury

Variations in the protein level of Omi/HtrA2 in the heart of aged rats may contribute to the increased susceptibility of cardiomyocytes to ischemia/reperfusion injury and cell death : Omi/HtrA2 and aged heart injury

Item
http://www.wikidata.org/entity/Q28583230
Mitochondrial Metabolism in Aging HeartMitochondrial quality control in the myocardium: cooperation between protein degradation and mitophagyCloning and Transcriptional Activity of the Mouse Omi/HtrA2 Gene Promoter.Thioredoxin reductase was nitrated in the aging heart after myocardial ischemia/reperfusion.Alleviation of hyperglycemia induced vascular endothelial injury by exenatide might be related to the reduction of nitrooxidative stress.Cardiac Specific Overexpression of Mitochondrial Omi/HtrA2 Induces Myocardial Apoptosis and Cardiac Dysfunction.Ischemia/Reperfusion.Responses of A549 human lung epithelial cells to cristobalite and α-quartz exposures assessed by toxicoproteomics and gene expression analysis.The protective effects of ischemic preconditioning on rats with renal ischemia-reperfusion injury and the effects on the expression of Bcl-2 and Bax.Phytoestrogen α-Zearalanol attenuates homocysteine-induced apoptosis in human umbilical vein endothelial cells.Impact of age on cardiovascular function, inflammation, and oxidative stress in experimental asphyxial cardiac arrest.A soluble receptor for advanced glycation end-products inhibits myocardial apoptosis induced by ischemia/reperfusion via the JAK2/STAT3 pathway.The mitochondria-targeting peptide elamipretide diminishes circulating HtrA2 in ST-segment elevation myocardial infarction.
P2860

Variations in the protein level of Omi/HtrA2 in the heart of aged rats may contribute to the increased susceptibility of cardiomyocytes to ischemia/reperfusion injury and cell death : Omi/HtrA2 and aged heart injury

Item
http://www.wikidata.org/entity/Q28583230
description
2013 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի հունիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2013
@ast
im Juni 2013 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2013/06/01)
@sk
vědecký článek publikovaný v roce 2013
@cs
wetenschappelijk artikel (gepubliceerd op 2013/06/01)
@nl
наукова стаття, опублікована в червні 2013
@uk
مقالة علمية (نشرت في يونيو 2013)
@ar
name
Variations in the protein leve ...... mi/HtrA2 and aged heart injury
@ast
Variations in the protein leve ...... mi/HtrA2 and aged heart injury
@en
Variations in the protein leve ...... mi/HtrA2 and aged heart injury
@nl
type
label
Variations in the protein leve ...... mi/HtrA2 and aged heart injury
@ast
Variations in the protein leve ...... mi/HtrA2 and aged heart injury
@en
Variations in the protein leve ...... mi/HtrA2 and aged heart injury
@nl
prefLabel
Variations in the protein leve ...... mi/HtrA2 and aged heart injury
@ast
Variations in the protein leve ...... mi/HtrA2 and aged heart injury
@en
Variations in the protein leve ...... mi/HtrA2 and aged heart injury
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P433
P478
P577
P698
P921
P932
P356
P1433
P1476
Variations in the protein leve ...... mi/HtrA2 and aged heart injury
@en
P2093
P2860
P304
P31
P356
P433
P478
P577
P698
P921
P932