Chemokine contribution to neuropathic pain: respective induction of CXCL1 and CXCR2 in spinal cord astrocytes and neurons.
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Hypersensitivity Responses in the Central Nervous SystemEmerging targets in neuroinflammation-driven chronic painInhibition of the spinal astrocytic JNK/MCP-1 pathway activation correlates with the analgesic effects of tanshinone IIA sulfonate in neuropathic painChemokine receptor CXCR4 regulates CaMKII/CREB pathway in spinal neurons that underlies cancer-induced bone pain.Astrocyte Activation in Locus Coeruleus Is Involved in Neuropathic Pain Exacerbation Mediated by Maternal Separation and Social Isolation Stress.Pathological pain and the neuroimmune interface.Connexin-43 induces chemokine release from spinal cord astrocytes to maintain late-phase neuropathic pain in mice.Neutrophil contribution to spinal cord injury and repair.Chemokine CXCL1 enhances inflammatory pain and increases NMDA receptor activity and COX-2 expression in spinal cord neurons via activation of CXCR2.TRAF6 upregulation in spinal astrocytes maintains neuropathic pain by integrating TNF-α and IL-1β signalingExperimental autoimmune prostatitis induces microglial activation in the spinal cord.Pharmacological inhibition of CXCR2 chemokine receptors modulates paraquat-induced intoxication in rats.Astrocytes Are Primed by Chronic Neurodegeneration to Produce Exaggerated Chemokine and Cell Infiltration Responses to Acute Stimulation with the Cytokines IL-1β and TNF-α.Identification of lncRNA expression profile in the spinal cord of mice following spinal nerve ligation-induced neuropathic pain.A new minimally-invasive method for microinjection into the mouse spinal dorsal horn.Bedding Material Affects Mechanical Thresholds, Heat Thresholds, and Texture PreferenceCXCL13 drives spinal astrocyte activation and neuropathic pain via CXCR5.Paeoniflorin and Albiflorin Attenuate Neuropathic Pain via MAPK Pathway in Chronic Constriction Injury Rats.Chemokine CXCL13 mediates orofacial neuropathic pain via CXCR5/ERK pathway in the trigeminal ganglion of miceOverexpression of GRK6 attenuates neuropathic pain via suppression of CXCR2 in rat dorsal root ganglion.Downregulation of miR-219 enhances brain-derived neurotrophic factor production in mouse dorsal root ganglia to mediate morphine analgesic tolerance by upregulating CaMKIIγ.Effect of pioglitazone on neuropathic pain and spinal expression of TLR-4 and cytokines.CXCL13/CXCR5 enhances sodium channel Nav1.8 current density via p38 MAP kinase in primary sensory neurons following inflammatory painNFκB-mediated CXCL1 production in spinal cord astrocytes contributes to the maintenance of bone cancer pain in mice.CXCL12 in astrocytes contributes to bone cancer pain through CXCR4-mediated neuronal sensitization and glial activation in rat spinal cord.Blockade of CXCR2 signalling: a potential therapeutic target for preventing neutrophil-mediated inflammatory diseases.JNK in spinal cord facilitates bone cancer pain in rats through modulation of CXCL1.Chemokine Receptor CXCR3 in the Spinal Cord Contributes to Chronic Itch in Mice.Chemokine CXCL13 activates p38 MAPK in the trigeminal ganglion after infraorbital nerve injury.The immunopathogenesis of seropositive rheumatoid arthritis: from triggering to targeting.Prenatal alcohol exposure potentiates chronic neuropathic pain, spinal glial and immune cell activation and alters sciatic nerve and DRG cytokine levels.Chemokines in neuron-glial cell interaction and pathogenesis of neuropathic pain.Calcitonin gene-related peptide contributes to peripheral nerve injury-induced mechanical hypersensitivity through CCL5 and p38 pathwaysAutoantibodies to citrullinated proteins induce joint pain independent of inflammation via a chemokine-dependent mechanism.Upregulation of Chemokine CXCL12 in the Dorsal Root Ganglia and Spinal Cord Contributes to the Development and Maintenance of Neuropathic Pain Following Spared Nerve Injury in Rats.[EXPRESS] The Chemokine Receptor CXCR2 Supports Nociceptive Sensitization after Traumatic Brain Injury.The role of some chemokines from the CXC subfamily in a mouse model of diabetic neuropathyTNF-α/TNFR1 Signaling is Required for the Full Expression of Acute and Chronic Itch in Mice via Peripheral and Central Mechanisms.Opioids enhance CXCL1 expression and function after incision in mice.LncRNA expression in the spinal cord modulated by minocycline in a mouse model of spared nerve injury.
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Chemokine contribution to neuropathic pain: respective induction of CXCL1 and CXCR2 in spinal cord astrocytes and neurons.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on 04 July 2013
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Chemokine contribution to neur ...... l cord astrocytes and neurons.
@en
Chemokine contribution to neur ...... l cord astrocytes and neurons.
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type
label
Chemokine contribution to neur ...... l cord astrocytes and neurons.
@en
Chemokine contribution to neur ...... l cord astrocytes and neurons.
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prefLabel
Chemokine contribution to neur ...... l cord astrocytes and neurons.
@en
Chemokine contribution to neur ...... l cord astrocytes and neurons.
@nl
P2093
P2860
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Chemokine contribution to neur ...... l cord astrocytes and neurons.
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P2093
Zhi-Jun Zhang
P2860
P304
P356
10.1016/J.PAIN.2013.07.002
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P577
2013-07-04T00:00:00Z