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Arrhythmogenic KCNE gene variants: current knowledge and future challenges.

Arrhythmogenic KCNE gene variants: current knowledge and future challenges.

http://www.wikidata.org/entity/Q37511443

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The KCNE2 K+ channel regulatory subunit: Ubiquitous influence, complex pathobiology Kcne4 deletion sex- and age-specifically impairs cardiac repolarization in mice.Novel exon 1 protein-coding regions N-terminally extend human KCNE3 and KCNE4.KCNE1 and KCNE3: The yin and yang of voltage-gated K(+) channel regulation Regulation of human cardiac potassium channels by full-length KCNE3 and KCNE4.KCNE4 and KCNE5: K(+) channel regulation and cardiac arrhythmogenesis.Transmural gradients in ion channel and auxiliary subunit expression.A snapshot of genetic and epigenetic basis of arrhythmia and heart failure.Potassium channels in the heart: structure, function and regulation.High-throughput genetic characterization of a cohort of Brugada syndrome patients

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Arrhythmogenic KCNE gene variants: current knowledge and future challenges.

http://www.wikidata.org/entity/Q37511443

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 24 January 2014

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Arrhythmogenic KCNE gene variants: current knowledge and future challenges.

@en

Arrhythmogenic KCNE gene variants: current knowledge and future challenges.

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type

label

Arrhythmogenic KCNE gene variants: current knowledge and future challenges.

@en

Arrhythmogenic KCNE gene variants: current knowledge and future challenges.

@nl

prefLabel

Arrhythmogenic KCNE gene variants: current knowledge and future challenges.

@en

Arrhythmogenic KCNE gene variants: current knowledge and future challenges.

@nl

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FGENE.2014.00003

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Frontiers in Genetics

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Arrhythmogenic KCNE gene variants: current knowledge and future challenges.

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Geoffrey W Abbott

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Shawn M Crump

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P2860

Kcne2 deletion uncovers its crucial role in thyroid hormone biosynthesis

MiRP1 forms IKr potassium channels with HERG and is associated with cardiac arrhythmia

Cellular dysfunction of LQT5-minK mutants: abnormalities of IKs, IKr and trafficking in long QT syndrome

A constitutively open potassium channel formed by KCNQ1 and KCNE3

KCNE2 confers background current characteristics to the cardiac KCNQ1 potassium channel

KCNE5 (KCNE1L) variants are novel modulators of Brugada syndrome and idiopathic ventricular fibrillation

KCNE2 protein is more abundant in ventricles than in atria and can accelerate hERG protein degradation in a phosphorylation-dependent manner

KCNE5 induces time- and voltage-dependent modulation of the KCNQ1 current.

Regulation of the Kv2.1 potassium channel by MinK and MiRP1

Positional cloning of a novel potassium channel gene: KVLQT1 mutations cause cardiac arrhythmias

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3

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scholarly article

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10.3389/FGENE.2014.00003

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5

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2014-01-24T00:00:00Z

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259986655

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24478792

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3900874

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