Potential role of the src gene product in inhibition of gap-junctional communication in NIH/3T3 cells
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Dissection of the molecular basis of pp60(v-src) induced gating of connexin 43 gap junction channels.Aberrant expression and function of gap junctions during carcinogenesis.Immunolocalization of the cellular src protein in interphase and mitotic NIH c-src overexpresser cells.5-Aza-2'-deoxycytidine suppresses human renal carcinoma cell growth in a xenograft model via up-regulation of the connexin 32 gene.Induction of sensitivity to the cytotoxic action of tumor necrosis factor alpha by adenovirus E1A is independent of transformation and transcriptional activationExpression of H-ras correlates with metastatic potential: evidence for direct regulation of the metastatic phenotype in 10T1/2 and NIH 3T3 cellsLoss of intercellular junctional communication correlates with metastatic potential in mammary adenocarcinoma cells.Intercellular communication in bronchial epithelial cells: review of evidence for a possible role in lung carcinogenesis.Connexin43 cardiac gap junction remodeling: lessons from genetically engineered murine models.An overview of tumor biology.Altered junctional permeability between cells transformed by v-ras, v-mos, or v-src.Connexin expression and cell coupling fail to reverse the v-src transformed growth characteristics of a Cx43-/- cell line.The tumor promoter 12-O-tetradecanoylphorbol-13-acetate and the ras oncogene modulate expression and phosphorylation of gap junction proteins.Tyrosine phosphorylation of the gap junction protein connexin43 is required for the pp60v-src-induced inhibition of communication.Phosphorylation of connexin43 gap junction protein in uninfected and Rous sarcoma virus-transformed mammalian fibroblasts.Interaction of c-Src with gap junction protein connexin-43. Role in the regulation of cell-cell communication.Tyrosine phosphorylation of connexin 43 by v-Src is mediated by SH2 and SH3 domain interactions.Early activation of endogenous pp60src kinase activity during neuronal differentiation of cultured human neuroblastoma cells.Herbimycin A suppresses the reduction of gap-junctional intercellular communication induced by tumor-promoting phorbol ester in 3T3-L1 cells.Tumor promoters enhance v-myc-induced focus formation in mammalian cell lines.
P2860
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P2860
Potential role of the src gene product in inhibition of gap-junctional communication in NIH/3T3 cells
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on August 1985
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Potential role of the src gene ...... communication in NIH/3T3 cells
@en
Potential role of the src gene ...... ommunication in NIH/3T3 cells.
@nl
type
label
Potential role of the src gene ...... communication in NIH/3T3 cells
@en
Potential role of the src gene ...... ommunication in NIH/3T3 cells.
@nl
prefLabel
Potential role of the src gene ...... communication in NIH/3T3 cells
@en
Potential role of the src gene ...... ommunication in NIH/3T3 cells.
@nl
P2093
P2860
P356
P1476
Potential role of the src gene ...... communication in NIH/3T3 cells
@en
P2093
P2860
P304
P356
10.1073/PNAS.82.16.5360
P407
P577
1985-08-01T00:00:00Z