A failure in energy metabolism and antioxidant uptake precede symptoms of Huntington's disease in mice. - Wikidata - wikimedia - TriplyDB

A failure in energy metabolism and antioxidant uptake precede symptoms of Huntington's disease in mice.

A failure in energy metabolism and antioxidant uptake precede symptoms of Huntington's disease in mice.

http://www.wikidata.org/entity/Q37528334

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Insights into the Pathology of the α2-Na(+)/K(+)-ATPase in Neurological Disorders; Lessons from Animal Models.Old Things New View: Ascorbic Acid Protects the Brain in Neurodegenerative Disorders Vitamin C Transporters in Cancer: Current Understanding and Gaps in Knowledge.How Energy Metabolism Supports Cerebral Function: Insights from 13C Magnetic Resonance Studies In vivo Impaired brain energy metabolism in the BACHD mouse model of Huntington's disease: critical role of astrocyte-neuron interactions.miR-196a Ameliorates Cytotoxicity and Cellular Phenotype in Transgenic Huntington's Disease Monkey Neural Cells Beyond the redox imbalance: Oxidative stress contributes to an impaired GLUT3 modulation in Huntington's disease Integration of targeted metabolomics and transcriptomics identifies deregulation of phosphatidylcholine metabolism in Huntington's disease peripheral blood samples Vitamin C Transporters, Recycling and the Bystander Effect in the Nervous System: SVCT2 versus Gluts.SVCT2 Is Expressed by Cerebellar Precursor Cells, Which Differentiate into Neurons in Response to Ascorbic Acid.Principles of sodium homeostasis and sodium signalling in astroglia.Astrocytes are key but indirect contributors to the development of the symptomatology and pathophysiology of Huntington's disease.Ascorbic Acid to Manage Psychiatric Disorders.Does Vitamin C Influence Neurodegenerative Diseases and Psychiatric Disorders?Metabolic and transcriptomic analysis of Huntington's disease model reveal changes in intracellular glucose levels and related genes Reduced cell size, chromosomal aberration and altered proliferation rates are characteristics and confounding factors in the STHdh cell model of Huntington disease.Preventive and therapeutic potential of ascorbic acid in neurodegenerative diseases.Spatiotemporal Proteomic Profiling of Huntington's Disease Inclusions Reveals Widespread Loss of Protein Function.Towards an Understanding of Energy Impairment in Huntington's Disease Brain.Neuroprotective potential of astroglia.Dihydromyricetin Ameliorates 3NP-induced Behavioral Deficits and Striatal Injury in Rats.The Tiny for the Biggest Answers in Huntington's Disease

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A failure in energy metabolism and antioxidant uptake precede symptoms of Huntington's disease in mice.

http://www.wikidata.org/entity/Q37528334

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on January 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

A failure in energy metabolism ...... Huntington's disease in mice.

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A failure in energy metabolism ...... Huntington's disease in mice.

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type

label

A failure in energy metabolism ...... Huntington's disease in mice.

@en

A failure in energy metabolism ...... Huntington's disease in mice.

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prefLabel

A failure in energy metabolism ...... Huntington's disease in mice.

@en

A failure in energy metabolism ...... Huntington's disease in mice.

@nl

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Nature Communications

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A failure in energy metabolism ...... f Huntington's disease in mice

@en

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Alejandra V Parra

http://www.w3.org/2001/XMLSchema#string

Aníbal I Acuña

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Carlos Cepeda

http://www.w3.org/2001/XMLSchema#string

Carlos Kramm

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Claudio Hetz

http://www.w3.org/2001/XMLSchema#string

Felipe A Beltrán

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Ilona I Concha

http://www.w3.org/2001/XMLSchema#string

Magdalena Esparza

http://www.w3.org/2001/XMLSchema#string

Maite A Castro

http://www.w3.org/2001/XMLSchema#string

Michael S Levine

http://www.w3.org/2001/XMLSchema#string

P2860

Huntingtin phosphorylation acts as a molecular switch for anterograde/retrograde transport in neurons

Near-membrane dynamics and capture of TRPM8 channels within transient confinement domains

A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes.

Delivery of GABAARs to synapses is mediated by HAP1-KIF5 and disrupted by mutant huntingtin.

Molecular identification and functional characterization of the vitamin C transporters expressed by Sertoli cells

Interaction of Huntingtin-associated protein-1 with kinesin light chain: implications in intracellular trafficking in neurons

Huntingtin-HAP40 complex is a novel Rab5 effector that regulates early endosome motility and is up-regulated in Huntington's disease.

Huntingtin coordinates the dynein-mediated dynamic positioning of endosomes and lysosomes.

Sex differences in behavior and striatal ascorbate release in the 140 CAG knock-in mouse model of Huntington's disease.

Fluorescence recovery after photobleaching studies of lipid rafts.

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2917

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scholarly article

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10.1038/NCOMMS3917

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4

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Sebastian Brauchi

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2013-01-01T00:00:00Z

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259320644

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24336051

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Huntington disease

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3905737

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