Vaginal epithelial cell-derived S100 alarmins induced by Candida albicans via pattern recognition receptor interactions are sufficient but not necessary for the acute neutrophil response during experimental vaginal candidiasis.

Vaginal epithelial cell-derived S100 alarmins induced by Candida albicans via pattern recognition receptor interactions are sufficient but not necessary for the acute neutrophil response during experimental vaginal candidiasis.

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The Host's Reply to Candida Biofilm Candida vaginitis: when opportunism knocks, the host responds Upregulation of calprotectin in mild IgE-mediated ovalbumin hypersensitivity.Zinc and Manganese Chelation by Neutrophil S100A8/A9 (Calprotectin) Limits Extracellular Aspergillus fumigatus Hyphal Growth and Corneal Infection.Secretory Aspartyl Proteinases Cause Vaginitis and Can Mediate Vaginitis Caused by Candida albicans in Mice.Transcriptomic analysis of vulvovaginal candidiasis identifies a role for the NLRP3 inflammasome.A Candida albicans Strain Expressing Mammalian Interleukin-17A Results in Early Control of Fungal Growth during Disseminated Infection.Adaptive immune responses to Candida albicans infection.Candida albicans-epithelial interactions and pathogenicity mechanisms: scratching the surface.The role of pattern recognition receptors in the innate recognition of Candida albicans.Innate immune cell response upon Candida albicans infection Fungal morphogenetic pathways are required for the hallmark inflammatory response during Candida albicans vaginitis.Vaginal Heparan Sulfate Linked to Neutrophil Dysfunction in the Acute Inflammatory Response Associated with Experimental Vulvovaginal Candidiasis.Vulvovaginal candidosis: contemporary challenges and the future of prophylactic and therapeutic approaches.Contribution of TLR2 pathway in the pathogenesis of vulvovaginal candidiasis.The Absence of N-Acetyl-D-glucosamine Causes Attenuation of Virulence of Candida albicans upon Interaction with Vaginal Epithelial Cells In Vitro.The role of calprotectin in withholding zinc and copper from Candida albicans.Candidalysin drives epithelial signaling, neutrophil recruitment, and immunopathology at the vaginal mucosa.Innate Immunity to Mucosal Candida Infections.Immune Recognition of Fungal Polysaccharides.Experimental models of vaginal candidiasis and inflammation.A novel mechanism behind the immunopathogenesis of vulvovaginal candidiasis: "Neutrophil Anergy".Candida Immunity

P2860

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P2860

Vaginal epithelial cell-derived S100 alarmins induced by Candida albicans via pattern recognition receptor interactions are sufficient but not necessary for the acute neutrophil response during experimental vaginal candidiasis.

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http://www.wikidata.org/entity/Q37546318

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article científic

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bilimsel makale

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scientific article published on 09 December 2013

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