The herpes simplex virus type 1 latency-associated transcript (LAT) enhancer/rcr is hyperacetylated during latency independently of LAT transcription
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Herpesviral latency-associated transcript gene promotes assembly of heterochromatin on viral lytic-gene promoters in latent infection.A chromatin insulator-like element in the herpes simplex virus type 1 latency-associated transcript region binds CCCTC-binding factor and displays enhancer-blocking and silencing activities.CTCF-dependent chromatin boundary element between the latency-associated transcript and ICP0 promoters in the herpes simplex virus type 1 genomeHerpes simplex virus type 1 latency and reactivation: an updateA comparison of herpes simplex virus type 1 and varicella-zoster virus latency and reactivationThe molecular basis of herpes simplex virus latencyTargeting CTCF to Control Virus Gene Expression: A Common Theme amongst Diverse DNA VirusesGenital Herpes: Insights into Sexually Transmitted Infectious DiseaseRole of chromatin during herpesvirus infections.Herpes simplex virus VP16, but not ICP0, is required to reduce histone occupancy and enhance histone acetylation on viral genomes in U2OS osteosarcoma cells.Control of alpha-herpesvirus IE gene expression by HCF-1 coupled chromatin modification activities.Epigenetic regulation of latent HSV-1 gene expression.Changes to euchromatin on LAT and ICP4 following reactivation are more prevalent in an efficiently reactivating strain of HSV-1.Common threads in persistent viral infectionsRegulation of herpes simplex virus type 1 thymidine kinase gene expression by thyroid hormone receptor in cultured neuronal cells.Snapshots: chromatin control of viral infection.Deacetylation of the herpes simplex virus type 1 latency-associated transcript (LAT) enhancer and a decrease in LAT abundance precede an increase in ICP0 transcriptional permissiveness at early times postexplant.Epigenetic regulation of varicella-zoster virus open reading frames 62 and 63 in latently infected human trigeminal ganglia.Tegument protein control of latent herpesvirus establishment and animation.Transcription of the herpes simplex virus latency-associated transcript promotes the formation of facultative heterochromatin on lytic promoters.Upregulation of mouse genes in HSV-1 latent TG after butyrate treatment implicates the multiple roles of the LAT-ICP0 locus.Herpes simplex virus type 1 latently infected neurons differentially express latency-associated and ICP0 transcripts.Herpes simplex virus 2 microRNA miR-H6 is a novel latency-associated transcript-associated microRNA, but reduction of its expression does not influence the establishment of viral latency or the recurrence phenotype.Tissue-specific splicing of the herpes simplex virus type 1 latency-associated transcript (LAT) intron in LAT transgenic miceReview: The neurobiology of varicella zoster virus infection.Characterization of herpes simplex virus 2 primary microRNA Transcript regulationDynamic modulation of HSV chromatin drives initiation of infection and provides targets for epigenetic therapies.Nuclear sensing of viral DNA, epigenetic regulation of herpes simplex virus infection, and innate immunityThe number of alphaherpesvirus particles infecting axons and the axonal protein repertoire determines the outcome of neuronal infectionSodium butyrate: a chemical inducer of in vivo reactivation of herpes simplex virus type 1 in the ocular mouse model.The HSV-1 Latency-Associated Transcript Functions to Repress Latent Phase Lytic Gene Expression and Suppress Virus Reactivation from Latently Infected NeuronsReplication-Competent Controlled Herpes Simplex Virus.In vivo changes in the patterns of chromatin structure associated with the latent herpes simplex virus type 1 genome in mouse trigeminal ganglia can be detected at early times after butyrate treatment.CTCF occupation of the herpes simplex virus 1 genome is disrupted at early times postreactivation in a transcription-dependent manner.A novel selective LSD1/KDM1A inhibitor epigenetically blocks herpes simplex virus lytic replication and reactivation from latency.Reduction in severity of a herpes simplex virus type 1 murine infection by treatment with a ribozyme targeting the UL20 gene RNA.Association of the cellular coactivator HCF-1 with the Golgi apparatus in sensory neurons.A Herpesviral Lytic Protein Regulates the Structure of Latent Viral Chromatin.The dynamics of HCF-1 modulation of herpes simplex virus chromatin during initiation of infection.Chromatin control of herpes simplex virus lytic and latent infection.
P2860
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P2860
The herpes simplex virus type 1 latency-associated transcript (LAT) enhancer/rcr is hyperacetylated during latency independently of LAT transcription
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
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2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
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name
The herpes simplex virus type ...... pendently of LAT transcription
@en
The herpes simplex virus type 1 latency-associated transcript
@nl
type
label
The herpes simplex virus type ...... pendently of LAT transcription
@en
The herpes simplex virus type 1 latency-associated transcript
@nl
prefLabel
The herpes simplex virus type ...... pendently of LAT transcription
@en
The herpes simplex virus type 1 latency-associated transcript
@nl
P2093
P2860
P1433
P1476
The herpes simplex virus type ...... pendently of LAT transcription
@en
P2093
Antonio L Amelio
David C Bloom
Nicole J Kubat
Nicole V Giordani
P2860
P304
12508-12518
P356
10.1128/JVI.78.22.12508-12518.2004
P407
P577
2004-11-01T00:00:00Z