The paradox of NKp46+ natural killer cells: drivers of severe hepatitis C virus-induced pathology but in-vivo resistance to interferon α treatment.
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Natural killer cells in hepatitis C: Current progressHepatitis C Virus and Human Cytomegalovirus-Natural Killer Cell Subsets in Persistent Viral Infections.Chronic HCV infection affects the NK cell phenotype in the blood more than in the liver.NK cells and their ability to modulate T cells during virus infectionsNK cell activation in human hantavirus infection explained by virus-induced IL-15/IL15Rα expression.Tracking the kinetics of intrahepatic immune responses by repeated fine needle aspiration of the liver.Interferon Alpha Induces Sustained Changes in NK Cell Responsiveness to Hepatitis B Viral Load Suppression In Vivo.Accumulation of Intrahepatic TNF-α-Producing NKp44+ NK Cells Correlates With Liver Fibrosis and Viral Load in Chronic HCV InfectionThe natural killer cell response to HCV infection.Between Scylla and Charybdis: the role of the human immune system in the pathogenesis of hepatitis C.Transcriptional Network Architecture of Breast Cancer Molecular SubtypesSTAT4-associated natural killer cell tolerance following liver transplantation.Natural cytotoxicity receptor-dependent natural killer cytolytic activity directed at hepatitis C Virus (HCV) is associated with liver inflammation, African American race, IL28B genotype, and response to pegylated interferon/ribavirin therapy in chrNatural killer cell heterogeneity: cellular dysfunction and significance in HIV-1 immuno-pathogenesis.Impact of natural killer cells on chronic hepatitis C and hepatocellular carcinoma.Immune regulation in chronic hepatitis C virus infection.Chronic infections with viruses or parasites: breaking bad to make good.Current progress in host innate and adaptive immunity against hepatitis C virus infection.NK cells in self-limited HCV infection exhibit a more extensively differentiated, but not memory-like, repertoire.Non-neutralizing epitopes induce robust hepatitis C virus (HCV)-specific antibody-dependent CD56+ natural killer cell responses in chronic HCV-infected patients.Spontaneous and natural cytotoxicity receptor-mediated cytotoxicity are effector functions of distinct natural killer subsets in hepatitis C virus-infected chimpanzees.Lack of Siglec-7 expression identifies a dysfunctional natural killer cell subset associated with liver inflammation and fibrosis in chronic HCV infection.Increased expressions of NKp44, NKp46 on NK/NKT-like cells are associated with impaired cytolytic function in self-limiting hepatitis E infection.NKp46+ expression on NK cells as a biomarker for liver pathology and IFN-responiveness in HCV infection.Liver sampling: a vital window into HBV pathogenesis on the path to functional cure.IL-12 and IL-15 induce the expression of CXCR6 and CD49a on peripheral natural killer cells.CEACAM1 Is Associated With the Suppression of Natural Killer Cell Function in Patients With Chronic Hepatitis CTNFR2/BIRC3-TRAF1 signaling pathway as a novel NK cell immune checkpoint in cancer
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The paradox of NKp46+ natural killer cells: drivers of severe hepatitis C virus-induced pathology but in-vivo resistance to interferon α treatment.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
@tr
scientific article published on 11 May 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
@cs
name
The paradox of NKp46+ natural ...... nce to interferon α treatment.
@en
The paradox of NKp46+ natural ...... nce to interferon α treatment.
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type
label
The paradox of NKp46+ natural ...... nce to interferon α treatment.
@en
The paradox of NKp46+ natural ...... nce to interferon α treatment.
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prefLabel
The paradox of NKp46+ natural ...... nce to interferon α treatment.
@en
The paradox of NKp46+ natural ...... nce to interferon α treatment.
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P2860
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The paradox of NKp46+ natural ...... ance to interferon α treatment
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P2093
Adam Christian
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P304
P356
10.1136/GUTJNL-2013-304472
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P577
2013-05-11T00:00:00Z