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Inflammatory consequences in a rodent model of mild traumatic brain injury.

Inflammatory consequences in a rodent model of mild traumatic brain injury.

http://www.wikidata.org/entity/Q37616240

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Bryostatin-1 Restores Blood Brain Barrier Integrity following Blast-Induced Traumatic Brain Injury Long-lasting suppression of acoustic startle response after mild traumatic brain injury.Effects of antioxidant treatment on blast-induced brain injury.Merging pathology with biomechanics using CHIMERA (Closed-Head Impact Model of Engineered Rotational Acceleration): a novel, surgery-free model of traumatic brain injury.Traumatic white matter injury and glial activation: from basic science to clinics.Stress-dose hydrocortisone reduces critical illness-related corticosteroid insufficiency associated with severe traumatic brain injury in rats.Chronic post-traumatic headache: clinical findings and possible mechanisms.Deficient pain modulatory systems in patients with mild traumatic brain and chronic post-traumatic headache: implications for its mechanism Traumatic brain injury induces neuroinflammation and neuronal degeneration that is associated with escalated alcohol self-administration in rats Genetic activation of mTORC1 signaling worsens neurocognitive outcome after traumatic brain injury.Motor, visual and emotional deficits in mice after closed-head mild traumatic brain injury are alleviated by the novel CB2 inverse agonist SMM-189 The Involvement of Pial Microvessels in Leukocyte Invasion after Mild Traumatic Brain Injury Emergence of cognitive deficits after mild traumatic brain injury due to hyperthermia.Etifoxine improves sensorimotor deficits and reduces glial activation, neuronal degeneration, and neuroinflammation in a rat model of traumatic brain injury.Mild Traumatic Brain Injury Produces Neuron Loss That Can Be Rescued by Modulating Microglial Activation Using a CB2 Receptor Inverse Agonist.Risk of Coronary Artery Disease in Patients With Traumatic Intracranial Hemorrhage: A Nationwide, Population-Based Cohort Study.Neuroinflammation in animal models of traumatic brain injury.Blast exposure causes dynamic microglial/macrophage responses and microdomains of brain microvessel dysfunction.Blood-brain barrier and traumatic brain injury.Impacts of stellate ganglion block on plasma NF-κB and inflammatory factors of TBI patients.Serotonergic hyperactivity as a potential factor in developmental, acquired and drug-induced synesthesia.Inflammatory cytokine receptor blockade in a rodent model of mild traumatic brain injury.Study of neurometabolic and behavioral alterations in rodent model of mild traumatic brain injury: a pilot study.Hyperthermia and Mild Traumatic Brain Injury: Effects on Inflammation and the Cerebral Vasculature.A rodent model of mild traumatic brain blast injury.Mood symptoms correlate with kynurenine pathway metabolites following sports-related concussion.

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P2860

Inflammatory consequences in a rodent model of mild traumatic brain injury.

http://www.wikidata.org/entity/Q37616240

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 06 May 2013

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Inflammatory consequences in a rodent model of mild traumatic brain injury.

@en

Inflammatory consequences in a rodent model of mild traumatic brain injury.

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type

label

Inflammatory consequences in a rodent model of mild traumatic brain injury.

@en

Inflammatory consequences in a rodent model of mild traumatic brain injury.

@nl

prefLabel

Inflammatory consequences in a rodent model of mild traumatic brain injury.

@en

Inflammatory consequences in a rodent model of mild traumatic brain injury.

@nl

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Journal of Neurotrauma

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Inflammatory consequences in a rodent model of mild traumatic brain injury.

@en

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Claire E Hulsebosch

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Douglas S Dewitt

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Geda C Unabia

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Guojing Xu

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Harriet C Rea

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J Regino Perez-Polo

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Kathia M Johnson

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Margaret A Parsley

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Smitha K Infante

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P2860

Endogenous expression of interleukin-4 regulates macrophage activation and confines cavity formation after traumatic spinal cord injury

Glutamate-induced losses of oligodendrocytes and neurons and activation of caspase-3 in the rat spinal cord

VEGF and VEGF receptor expression after experimental brain contusion in rat

Tumor necrosis factor-α synthesis inhibitor, 3,6'-dithiothalidomide, reverses behavioral impairments induced by minimal traumatic brain injury in mice

Extracellular superoxide concentration increases following cerebral hypoxia but does not affect cerebral blood flow.

Temporary disruption of the rat blood-brain barrier with a monoclonal antibody: a novel method for dynamic manganese-enhanced MRI

Experimental models of brain trauma.

Transgenic inhibition of astroglial NF-kappa B leads to increased axonal sparing and sprouting following spinal cord injury.

Dual role of tumor necrosis factor alpha in brain injury.

Experimental brain injury induces differential expression of tumor necrosis factor-alpha mRNA in the CNS.

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727-740

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scholarly article

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10.1089/NEU.2012.2650

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9

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30

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2013-05-06T00:00:00Z

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23360201

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traumatic brain injury

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3941841

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