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Immune activation in brain aging and neurodegeneration: too much or too little?

Immune activation in brain aging and neurodegeneration: too much or too little?

http://www.wikidata.org/entity/Q37617634

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The endocannabinoid system in normal and pathological brain ageing The ageing systemic milieu negatively regulates neurogenesis and cognitive function.NLRP3 is activated in Alzheimer's disease and contributes to pathology in APP/PS1 mice Targeting innate immunity for neurodegenerative disorders of the central nervous system Role of docosahexaenoic acid in the modulation of glial cells in Alzheimer's disease Exposure to general anesthesia and the risk of dementia Elusive roles for reactive astrocytes in neurodegenerative diseases Role of pro-inflammatory cytokines released from microglia in Alzheimer's disease Clearance systems in the brain-implications for Alzheimer disease Microglia in the outer retina and their relevance to pathogenesis of age-related macular degeneration Neuroprotection for traumatic brain injury Potential mechanisms of progranulin-deficient FTLD Inflammation and α-synuclein's prion-like behavior in Parkinson's disease--is there a link?The dopaminergic system in autoimmune diseases CD4 T cells in immunity and immunotherapy of Alzheimer's disease Microglial priming and enhanced reactivity to secondary insult in aging, and traumatic CNS injury, and neurodegenerative disease Neuroimmunological processes in Parkinson's disease and their relation to α-synuclein: microglia as the referee between neuronal processes and peripheral immunity Reduced β-amyloid pathology in an APP transgenic mouse model of Alzheimer's disease lacking functional B and T cells APP intracellular domain impairs adult neurogenesis in transgenic mice by inducing neuroinflammation Microglial interactions with synapses are modulated by visual experience Pyrrolidine Dithiocarbamate Prevents Neuroinflammation and Cognitive Dysfunction after Endotoxemia in Rats.AAV-dominant negative tumor necrosis factor (DN-TNF) gene transfer to the striatum does not rescue medium spiny neurons in the YAC128 mouse model of Huntington's disease The Indispensable Roles of Microglia and Astrocytes during Brain Development Ageing, neurodegeneration and brain rejuvenation Dietary Phytochemicals in Neuroimmunoaging: A New Therapeutic Possibility for Humans?Friend or foe: the dichotomous impact of T cells on neuro-de/re-generation during aging Isothiocyanates Are Promising Compounds against Oxidative Stress, Neuroinflammation and Cell Death that May Benefit Neurodegeneration in Parkinson's Disease Lipopolysaccharide from Rhodobacter sphaeroides Attenuates Microglia-Mediated Inflammation and Phagocytosis and Directs Regulatory T Cell Response P2Y receptors on astrocytes and microglia mediate opposite effects in astroglial proliferation Differential effects of aging and sex on stroke induced inflammation across the lifespan Suppression of neuroinflammation by astrocytic dopamine D2 receptors via αB-crystallin Isoflurane attenuates mouse microglial engulfment induced by lipopolysaccharide and interferon-γ possibly by inhibition of p38 mitogen-activated protein kinase.Learning and memory ... and the immune system.A neurodegenerative disease mutation that accelerates the clearance of apoptotic cells Role of CB1 cannabinoid receptors on GABAergic neurons in brain aging Prolonged oral cannabinoid administration prevents neuroinflammation, lowers β-amyloid levels and improves cognitive performance in Tg APP 2576 mice.Parallel declines in cognition, motivation, and locomotion in aging mice: association with immune gene upregulation in the medial prefrontal cortex Hypothalamic programming of systemic ageing involving IKK-β, NF-κB and GnRH Microglial beclin 1 regulates retromer trafficking and phagocytosis and is impaired in Alzheimer's disease.Early microglia activation in a mouse model of chronic glaucoma.

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Immune activation in brain aging and neurodegeneration: too much or too little?

http://www.wikidata.org/entity/Q37617634

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article científic

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on October 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Immune activation in brain aging and neurodegeneration: too much or too little?

@en

Immune activation in brain aging and neurodegeneration: too much or too little?

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type

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Immune activation in brain aging and neurodegeneration: too much or too little?

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Immune activation in brain aging and neurodegeneration: too much or too little?

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prefLabel

Immune activation in brain aging and neurodegeneration: too much or too little?

@en

Immune activation in brain aging and neurodegeneration: too much or too little?

@nl

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J.NEURON.2009.08.039

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Immune activation in brain aging and neurodegeneration: too much or too little?

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Kurt M Lucin

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Tony Wyss-Coray

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P2860

Mice deficient in TNF receptors are protected against dopaminergic neurotoxicity: implications for Parkinson's disease

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p62/SQSTM1 binds directly to Atg8/LC3 to facilitate degradation of ubiquitinated protein aggregates by autophagy

Protective effects of NSAIDs on the development of Alzheimer disease

Functional requirement for class I MHC in CNS development and plasticity

Innate immunity and transcription of MGAT-III and Toll-like receptors in Alzheimer's disease patients are improved by bisdemethoxycurcumin

Resting microglial cells are highly dynamic surveillants of brain parenchyma in vivo

Innate immune recognition

Inflammation and Alzheimer's disease

The cerebral proteopathies: neurodegenerative disorders of protein conformation and assembly

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scholarly article

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10.1016/J.NEURON.2009.08.039

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