The niacin/butyrate receptor GPR109A suppresses mammary tumorigenesis by inhibiting cell survival.
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Mucosal Interactions between Genetics, Diet, and Microbiome in Inflammatory Bowel DiseaseDietary metabolites and the gut microbiota: an alternative approach to control inflammatory and autoimmune diseasesThree measurable and modifiable enteric microbial biotransformations relevant to cancer prevention and treatment.Microbiota modulation of myeloid cells in cancer therapyThe role of the microbiota in inflammation, carcinogenesis, and cancer therapy.DNMT1 is essential for mammary and cancer stem cell maintenance and tumorigenesis.Hydroxycarboxylic acid receptors are essential for breast cancer cells to control their lipid/fatty acid metabolismEnterotype May Drive the Dietary-Associated Cardiometabolic Risk FactorsFrom microbe to man: the role of microbial short chain fatty acid metabolites in host cell biology.Niacin as antidyslipidemic drug.Molecular mechanisms of target recognition by lipid GPCRs: relevance for cancer.Ameliorative effects of pyrazinoic acid against oxidative and metabolic stress manifested in rats with dimethylhydrazine induced colonic carcinoma.Expression of fatty acid sensing G-protein coupled receptors in peripartal Holstein cows.Benefits of short-chain fatty acids and their receptors in inflammation and carcinogenesis.Gut Microbiota, Inflammation, and Colorectal Cancer.Cell-surface G-protein-coupled receptors for tumor-associated metabolites: A direct link to mitochondrial dysfunction in cancer.Marketed drugs used for the management of hypercholesterolemia as anticancer armament.Transcriptome analysis of G protein-coupled receptors in distinct genetic subgroups of acute myeloid leukemia: identification of potential disease-specific targets.Impacts of microbiome metabolites on immune regulation and autoimmunity.Human Gut Microbiota and Gastrointestinal Cancer.Sodium Butyrate Inhibits Inflammation and Maintains Epithelium Barrier Integrity in a TNBS-induced Inflammatory Bowel Disease Mice Model.Dietary Associations with a Breast Cancer Risk Biomarker Depend on Menopause Status.Butyrate Suppresses the Proliferation of Colorectal Cancer Cells via Targeting Pyruvate Kinase M2 and Metabolic Reprogramming.
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The niacin/butyrate receptor GPR109A suppresses mammary tumorigenesis by inhibiting cell survival.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 26 December 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
The niacin/butyrate receptor G ...... s by inhibiting cell survival.
@en
The niacin/butyrate receptor G ...... s by inhibiting cell survival.
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type
label
The niacin/butyrate receptor G ...... s by inhibiting cell survival.
@en
The niacin/butyrate receptor G ...... s by inhibiting cell survival.
@nl
prefLabel
The niacin/butyrate receptor G ...... s by inhibiting cell survival.
@en
The niacin/butyrate receptor G ...... s by inhibiting cell survival.
@nl
P2093
P2860
P1433
P1476
The niacin/butyrate receptor G ...... is by inhibiting cell survival
@en
P2093
Muthusamy Thangaraju
Nagendra Singh
Pamela M Martin
Puttur D Prasad
Rajneesh Pathania
Ravi N Padia
Sabarish Ramachandran
Sudha Ananth
Vadivel Ganapathy
P2860
P304
P356
10.1158/0008-5472.CAN-13-1451
P407
P577
2013-12-26T00:00:00Z