Skeletal muscle insulin resistance is the primary defect in type 2 diabetes.
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Lipid-Induced Insulin Resistance in Skeletal Muscle: The Chase for the Culprit Goes from Total Intramuscular Fat to Lipid Intermediates, and Finally to Species of Lipid IntermediatesCircadian clock regulation of skeletal muscle growth and repairMetabolic Inflammation-Differential Modulation by Dietary ConstituentsEffects of walking on low-grade inflammation and their implications for Type 2 DiabetesSkeletal Muscle Mitochondrial Bioenergetics and Morphology in High Fat Diet Induced Obesity and Insulin Resistance: Focus on Dietary Fat SourceInsulin resistance in clinical and experimental alcoholic liver diseaseNotch signaling as a novel regulator of metabolismExercise as an intervention to improve metabolic outcomes after intrauterine growth restrictionType 2 diabetes can be prevented with early pharmacological interventionThe functional significance of the skeletal muscle clock: lessons from Bmal1 knockout modelsPractical combination therapy based on pathophysiology of type 2 diabetesThe Metabolic Implications of Glucocorticoids in a High-Fat Diet Setting and the Counter-Effects of ExerciseHuman Skeletal Muscle Disuse Atrophy: Effects on Muscle Protein Synthesis, Breakdown, and Insulin Resistance-A Qualitative ReviewExercise and Regulation of Carbohydrate MetabolismRegulation of insulin sensitivity by serine/threonine phosphorylation of insulin receptor substrate proteins IRS1 and IRS2Regulation of glycogen synthase from mammalian skeletal muscle--a unifying view of allosteric and covalent regulationAnti-Diabetic Effects of Jiang Tang Xiao Ke Granule via PI3K/Akt Signalling Pathway in Type 2 Diabetes KKAy MiceSignaling of the p21-activated kinase (PAK1) coordinates insulin-stimulated actin remodeling and glucose uptake in skeletal muscle cells.Involvement of mTOR in Type 2 CRF Receptor Inhibition of Insulin Signaling in Muscle Cells.Vascular function, insulin action, and exercise: an intricate interplayAdiponectin and insulin cross talk: the microvascular connection.Exercise and type 2 diabetes: molecular mechanisms regulating glucose uptake in skeletal muscle.Prevention of antipsychotic-induced hyperglycaemia by vitamin D: a data mining prediction followed by experimental exploration of the molecular mechanism.Models of accelerated sarcopenia: critical pieces for solving the puzzle of age-related muscle atrophy.Insulin and metabolic stress stimulate multisite serine/threonine phosphorylation of insulin receptor substrate 1 and inhibit tyrosine phosphorylation.An evaluation of low volume high-intensity intermittent training (HIIT) for health risk reduction in overweight and obese menHepatocyte Growth Factor Prevented High-Fat Diet-Induced Obesity and Improved Insulin Resistance in Mice.Dissecting the role of AMP-activated protein kinase in human diseases.Type 2 diabetes and obesity induce similar transcriptional reprogramming in human myocytesSubcutaneous and Visceral Adipose Tissue Secretions from Extremely Obese Men and Women both Acutely Suppress Muscle Insulin SignalingQuercetin, a Lead Compound against Type 2 Diabetes Ameliorates Glucose Uptake via AMPK Pathway in Skeletal Muscle Cell Line.Type 2 diabetes increases and metformin reduces total, colorectal, liver and pancreatic cancer incidences in Taiwanese: a representative population prospective cohort study of 800,000 individuals.Whole-genome expression analyses of type 2 diabetes in human skin reveal altered immune function and burden of infection.Loss-of-function myostatin mutation increases insulin sensitivity and browning of white fat in Meishan pigs.Increasing effect of Tangzhiqing formula on IRS-1-dependent PI3K/AKT signaling in muscle.Multi-tissue computational modeling analyzes pathophysiology of type 2 diabetes in MKR mice.Exosomes participate in the alteration of muscle homeostasis during lipid-induced insulin resistance in mice.Alteration of microRNA expression correlates to fatty acid-mediated insulin resistance in mouse myoblasts.Towards the minimal amount of exercise for improving metabolic health: beneficial effects of reduced-exertion high-intensity interval training.Adipose depots, not disease-related factors, account for skeletal muscle insulin sensitivity in established and treated rheumatoid arthritis
P2860
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P2860
Skeletal muscle insulin resistance is the primary defect in type 2 diabetes.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
Skeletal muscle insulin resistance is the primary defect in type 2 diabetes.
@en
Skeletal muscle insulin resistance is the primary defect in type 2 diabetes.
@nl
type
label
Skeletal muscle insulin resistance is the primary defect in type 2 diabetes.
@en
Skeletal muscle insulin resistance is the primary defect in type 2 diabetes.
@nl
prefLabel
Skeletal muscle insulin resistance is the primary defect in type 2 diabetes.
@en
Skeletal muscle insulin resistance is the primary defect in type 2 diabetes.
@nl
P2860
P921
P356
P1433
P1476
Skeletal muscle insulin resistance is the primary defect in type 2 diabetes.
@en
P2093
Devjit Tripathy
P2860
P304
P356
10.2337/DC09-S302
P407
P478
32 Suppl 2
P5008
P577
2009-11-01T00:00:00Z