Inhibition of apoptosis by overexpressing Bcl-2 enhances gene amplification by a mechanism independent of aphidicolin pretreatment.
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Bcl-2 expression suppresses mismatch repair activity through inhibition of E2F transcriptional activityBcl-2 and Bcl-XL serve an anti-inflammatory function in endothelial cells through inhibition of NF-kappaB.Bcl-xL blocks activation of related adhesion focal tyrosine kinase/proline-rich tyrosine kinase 2 and stress-activated protein kinase/c-Jun N-terminal protein kinase in the cellular response to methylmethane sulfonate.Sp1 involvement in the 4beta-phorbol 12-myristate 13-acetate (TPA)-mediated increase in resistance to methotrexate in Chinese hamster ovary cells.
P2860
Inhibition of apoptosis by overexpressing Bcl-2 enhances gene amplification by a mechanism independent of aphidicolin pretreatment.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on April 1996
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Inhibition of apoptosis by ove ...... t of aphidicolin pretreatment.
@en
Inhibition of apoptosis by ove ...... t of aphidicolin pretreatment.
@nl
type
label
Inhibition of apoptosis by ove ...... t of aphidicolin pretreatment.
@en
Inhibition of apoptosis by ove ...... t of aphidicolin pretreatment.
@nl
prefLabel
Inhibition of apoptosis by ove ...... t of aphidicolin pretreatment.
@en
Inhibition of apoptosis by ove ...... t of aphidicolin pretreatment.
@nl
P2860
P356
P1476
Inhibition of apoptosis by ove ...... t of aphidicolin pretreatment.
@en
P2093
P2860
P304
P356
10.1073/PNAS.93.8.3394
P407
P577
1996-04-01T00:00:00Z