Evaluating effects of deferoxamine in a rat tibia critical bone defect model
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Dimethyloxalylglycine prevents bone loss in ovariectomized C57BL/6J mice through enhanced angiogenesis and osteogenesis.Stimulating angiogenesis mitigates the unloading-induced reduction in osteogenesis in early-stage bone repair in rats.Protective effect of salidroside against bone loss via hypoxia-inducible factor-1α pathway-induced angiogenesis.Deferoxamine released from poly(lactic-co-glycolic acid) promotes healing of osteoporotic bone defect via enhanced angiogenesis and osteogenesis.Bone substitute materials supplemented with prolyl hydroxylase inhibitors decrease osteoclastogenesisin vitro
P2860
Evaluating effects of deferoxamine in a rat tibia critical bone defect model
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 31 December 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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Evaluating effects of deferoxamine in a rat tibia critical bone defect model
@en
Evaluating effects of deferoxamine in a rat tibia critical bone defect model.
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type
label
Evaluating effects of deferoxamine in a rat tibia critical bone defect model
@en
Evaluating effects of deferoxamine in a rat tibia critical bone defect model.
@nl
prefLabel
Evaluating effects of deferoxamine in a rat tibia critical bone defect model
@en
Evaluating effects of deferoxamine in a rat tibia critical bone defect model.
@nl
P2093
P2860
P1476
Evaluating effects of deferoxamine in a rat tibia critical bone defect model
@en
P2093
Benjamin Keller
Bikramjit S Grewal
Laurence E Dahners
P2860
P356
10.1016/J.JOR.2013.12.005
P577
2013-12-31T00:00:00Z