Injury-induced HDAC5 nuclear export is essential for axon regeneration. - Wikidata - wikimedia - TriplyDB

Injury-induced HDAC5 nuclear export is essential for axon regeneration.

Injury-induced HDAC5 nuclear export is essential for axon regeneration.

http://www.wikidata.org/entity/Q37704511

about

What makes a RAG regeneration associated?Histone Deacetylases Exert Class-Specific Roles in Conditioning the Brain and Heart Against Acute Ischemic Injury Axonal transport: cargo-specific mechanisms of motility and regulation BMP signaling in axon regeneration HDAC signaling in neuronal development and axon regeneration Mitochondrial fission is an acute and adaptive response in injured motor neurons Targeting histone deacetylases: perspectives for epigenetic-based therapy in cardio-cerebrovascular disease Growth control mechanisms in neuronal regeneration The promise and perils of HDAC inhibitors in neurodegeneration Kinesin-1-powered microtubule sliding initiates axonal regeneration in Drosophila cultured neurons.Facilitation of axon regeneration by enhancing mitochondrial transport and rescuing energy deficits Effects of αTAT1 and HDAC5 on axonal regeneration in adult neurons.Syntaxin13 expression is regulated by mammalian target of rapamycin (mTOR) in injured neurons to promote axon regeneration Epigenetic memory: the Lamarckian brain The breadth and type of systemic inflammation and the risk of adverse neurological outcomes in extremely low gestation newborns.Post-translational modifications of tubulin: pathways to functional diversity of microtubules.Differential effect of HDAC3 on cytoplasmic and nuclear huntingtin aggregates.A surviving intact branch stabilizes remaining axon architecture after injury as revealed by in vivo imaging in the mouse spinal cord.Robust Axonal Regeneration Occurs in the Injured CAST/Ei Mouse CNS HLA-DRα1-mMOG-35-55 treatment of experimental autoimmune encephalomyelitis reduces CNS inflammation, enhances M2 macrophage frequency, and promotes neuroprotection.Protein deacetylases and axonal regeneration.Live Imaging of Calcium Dynamics during Axon Degeneration Reveals Two Functionally Distinct Phases of Calcium Influx.Histone acetylation inhibitors promote axon growth in adult dorsal root ganglia neurons HDAC4 is required for inflammation-associated thermal hypersensitivity.Filamin A is required in injured axons for HDAC5 activity and axon regeneration.Neuronal activity regulates remyelination via glutamate signalling to oligodendrocyte progenitors Activating Injury-Responsive Genes with Hypoxia Enhances Axon Regeneration through Neuronal HIF-1α.Ketamine produces antidepressant-like effects through phosphorylation-dependent nuclear export of histone deacetylase 5 (HDAC5) in rats.Both HDAC5 and HDAC6 are required for the proliferation and metastasis of melanoma cells.Exercise dependent increase in axon regeneration into peripheral nerve grafts by propriospinal but not sensory neurons after spinal cord injury is associated with modulation of regeneration-associated genes.Expression of class II HDACs in two mouse models of temporal lobe epilepsy.Signaling Over Distances.Molecular and Cellular Mechanisms of Axonal Regeneration After Spinal Cord Injury.RAS and downstream RAF-MEK and PI3K-AKT signaling in neuronal development, function and dysfunction Spinal cord reconstitution with homologous neural grafts enables robust corticospinal regeneration.An evolutionarily conserved mechanism for cAMP elicited axonal regeneration involves direct activation of the dual leucine zipper kinase DLK HDAC5, a potential therapeutic target and prognostic biomarker, promotes proliferation, invasion and migration in human breast cancer.Impaired histone deacetylases 5 and 6 expression mimics the effects of obesity and hypoxia on adipocyte function Gpr126/Adgrg6 Has Schwann Cell Autonomous and Nonautonomous Functions in Peripheral Nerve Injury and Repair.Epigenetic Regulation of Axon Regeneration after Neural Injury.

P2860

Q26795534-94D2CF15-AA84-41F1-AC07-474C952E359E Q26801660-7A92D21A-47B5-4673-A71D-0F4957FD2B79 Q26827748-2D0A92CB-2F1E-45DC-92AD-94925EA3B4F4 Q26852511-28412FB0-97B5-4609-8C40-AE553EEA2809 Q27013969-549B7959-1DF4-47F7-9583-F23E208D5928 Q27335694-C53B60CA-6534-402A-820E-0A6CFDD11007 Q28085588-9621FFB7-EC99-46A1-B076-27C6A9C5700E Q28086828-F48A9337-31F5-42A0-AFE9-534EEC1791F6 Q28087539-DCD5DCB5-4614-4673-80A9-32EC1AF2C616 Q30652986-EDA06EFC-B0FD-4236-8FA4-2E9CF49DD1E0 Q30780429-DE156A46-4E8B-4675-B73A-D6C4D305C258 Q33688273-A09A4DB7-61E4-4D09-AC99-2F6DE8ABC6AA Q34076051-298F9A2A-E06A-4A0D-8A02-B88067A8782D Q34323841-89199145-AF91-4A85-A5C7-5CD620F35977 Q34772323-CD7F006A-A843-424C-AA8C-E32F69C20B2B Q35132313-052E0AFE-0DA4-4F90-AEF2-108CB53A87C4 Q35403365-0DBEB670-6F51-49F8-8061-8C0D5D068202 Q35621213-0DEEB3B9-F380-40DF-8F74-E6A6F9CCA485 Q35690385-CA73A0B3-01F3-44B4-A9F6-A1F9B3A39546 Q35782781-4CC82373-EF7D-4FFD-9182-2A163FC82655 Q35837941-F9FD1338-7C2C-484A-9D40-92AAF38C8318 Q35838525-9B45A0AF-E1B4-4291-AFAE-0568EBEBD35F Q35853603-B81E8AF3-7537-410E-A85F-07523DAC2B77 Q35879002-6AF02441-07E7-4EB0-913F-AEF8D3301CB2 Q36050019-39FC250D-9DDA-44BD-99C2-5C8E4D120683 Q36145960-A7E8A80A-A24E-4017-AB90-E01DFBDD5F35 Q36304932-CA63EF50-FCF7-4751-ADED-71AFF00966FF Q36419883-3500D60A-4C27-422B-AB20-97C5E89F4B0B Q36446188-FA5784F9-A22F-40E3-8EC6-9B1DF3FAD2D4 Q36471367-A69B638C-2106-4186-8A08-12BA60806B37 Q36536614-A99E7943-96AD-4F8A-8093-7D4D2D945629 Q36539447-5A42C60E-8B0A-455C-B751-6C5903F23C65 Q36539451-B518C68A-F873-4906-9A03-43863D21E615 Q36580617-A696C4CC-8CAD-4E1A-9637-27C1ACD1BC86 Q36880076-C833EA11-588C-4801-BDD2-E5F8B1C44660 Q36978295-261A607C-9F8A-45DB-AD8C-449B2C880CFC Q37437368-1185D94C-8ACD-4F3B-9CF3-6E165E4F2721 Q37439991-7A1FEB79-9E63-4840-AF00-F77B673F02C7 Q37490348-9E5F6A04-5E89-4ED8-AAD1-B2246BF030A7 Q37637863-E614590E-6AB0-4B10-A3E7-C98B280E6AAC

P2860

Injury-induced HDAC5 nuclear export is essential for axon regeneration.

http://www.wikidata.org/entity/Q37704511

description

2013 nî lūn-bûn

@nan

2013年の論文

@ja

2013年学术文章

@wuu

2013年学术文章

@zh-cn

2013年学术文章

@zh-hans

2013年学术文章

@zh-my

2013年学术文章

@zh-sg

2013年學術文章

@yue

2013年學術文章

@zh

2013年學術文章

@zh-hant

name

Injury-induced HDAC5 nuclear export is essential for axon regeneration.

@en

Injury-induced HDAC5 nuclear export is essential for axon regeneration.

@nl

type

label

Injury-induced HDAC5 nuclear export is essential for axon regeneration.

@en

Injury-induced HDAC5 nuclear export is essential for axon regeneration.

@nl

prefLabel

Injury-induced HDAC5 nuclear export is essential for axon regeneration.

@en

Injury-induced HDAC5 nuclear export is essential for axon regeneration.

@nl

P1433

Q37704511-3CD202E7-E51E-47C2-A2D5-FD892DD3BF9E

P1476

Q37704511-EA35F137-D5F4-463B-80B2-B3F566639E3F

P2093

Q37704511-C7D94761-896F-43EF-A25C-4ECF40B0D3C3

Q37704511-D6074D12-07C5-445B-B054-E306BCDFADA4

P2860

Q37704511-0BF7CFE8-AFD4-4DBE-8734-6BB6056AA535

Q37704511-1A4AD2C4-A6AA-4651-A149-1B6463DD907C

Q37704511-1DF454E9-05A4-4C01-9258-25D8E3ED88BD

Q37704511-241BA10D-98A4-410A-99F1-737735D2DBD5

Q37704511-375B77DA-ADD7-4773-911E-3D79C44F0CEF

Q37704511-3883FF19-BB44-40A1-934E-4C209E254D27

Q37704511-452C15EC-CEF0-4454-8B4A-5295B1A89F15

Q37704511-49E18D67-B11A-4A19-9E7B-6C6CBFE927F9

Q37704511-4D5722E1-1739-4548-87BF-AD8E001CD798

Q37704511-4F63108B-1AEF-4812-9022-E283083F1CE1

P304

Q37704511-DA8E55E9-7C79-4BA1-A126-32E7E8AF1DE9

P31

Q37704511-CAC04097-A145-4CBE-A975-561F4F9A9A35

P356

Q37704511-9C297DAF-204F-4839-AA8E-70F36FFDE003

P407

Q37704511-E8CC66CA-6554-45C8-888E-B21D5AB4B282

P433

Q37704511-45208232-8005-4E4A-9F35-4D94ACAC28C8

P478

Q37704511-D5907701-D87D-49E7-8630-92FD8AFBA775

P50

Q37704511-16A69FDF-220E-446E-BD50-DA42D7E4D266

Q37704511-1E57F067-707D-4C12-8CB1-2268700755DC

P577

Q37704511-A7373ABF-981B-465A-ADCE-A7B7DD3B08C7

P5875

Q37704511-957D7785-ABA7-4A04-A28D-31F96A3FDAD8

P698

Q37704511-87A892AE-2B85-4D8A-BC84-283744C98C0D

P932

Q37704511-B9EED3BC-79D1-4B61-8571-E42353125479

P356

J.CELL.2013.10.004

P1433

P1476

Injury-induced HDAC5 nuclear export is essential for axon regeneration

@en

P2093

Valeria Cavalli

http://www.w3.org/2001/XMLSchema#string

Yongcheol Cho

http://www.w3.org/2001/XMLSchema#string

P2860

Enzymatic activity associated with class II HDACs is dependent on a multiprotein complex containing HDAC3 and SMRT/N-CoR

Protein kinases C and D mediate agonist-dependent cardiac hypertrophy through nuclear export of histone deacetylase 5

Folate regulation of axonal regeneration in the rodent central nervous system through DNA methylation

HDAC inhibition promotes neuronal outgrowth and counteracts growth cone collapse through CBP/p300 and P/CAF-dependent p53 acetylation

Histone deacetylases 5 and 9 govern responsiveness of the heart to a subset of stress signals and play redundant roles in heart development

Imaging neuronal subsets in transgenic mice expressing multiple spectral variants of GFP

Deficits in axonal transport precede ALS symptoms in vivo.

Subcellular knockout of importin β1 perturbs axonal retrograde signaling.

Nerve growth factor antiserum induces axotomy-like changes in neuropeptide expression in intact sympathetic and sensory neurons.

Calcium and cyclic AMP promote axonal regeneration in Caenorhabditis elegans and require DLK-1 kinase

P304

894-908

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1016/J.CELL.2013.10.004

http://www.w3.org/2001/XMLSchema#string

P407

P433

4

http://www.w3.org/2001/XMLSchema#string

P478

155

http://www.w3.org/2001/XMLSchema#string

P50

P577

2013-11-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

258426331

http://www.w3.org/2001/XMLSchema#string

P698

24209626

http://www.w3.org/2001/XMLSchema#string

P932

3987749

http://www.w3.org/2001/XMLSchema#string