The antitumor agent imexon activates antioxidant gene expression: evidence for an oxidative stress response. - Wikidata - wikimedia - TriplyDB

The antitumor agent imexon activates antioxidant gene expression: evidence for an oxidative stress response.

The antitumor agent imexon activates antioxidant gene expression: evidence for an oxidative stress response.

http://www.wikidata.org/entity/Q37707959

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Redox-directed cancer therapeutics: molecular mechanisms and opportunities Redox Homeostasis and Cellular Antioxidant Systems: Crucial Players in Cancer Growth and Therapy Anti-cancer effect of thiacremonone through down regulation of peroxiredoxin 6 Nrf2-dependent sulfiredoxin-1 expression protects against cigarette smoke-induced oxidative stress in lungs.Mechanistic and kinetic details of catalysis of thiol-disulfide exchange by glutaredoxins and potential mechanisms of regulation Expression of ABCG2 (BCRP) is regulated by Nrf2 in cancer cells that confers side population and chemoresistance phenotype.Mitophagy or how to control the Jekyll and Hyde embedded in mitochondrial metabolism: implications for melanoma progression and drug resistance.Oxidative stress and proteasome inhibitors in multiple myeloma.

P2860

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P2860

The antitumor agent imexon activates antioxidant gene expression: evidence for an oxidative stress response.

http://www.wikidata.org/entity/Q37707959

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on June 2007

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

The antitumor agent imexon act ...... an oxidative stress response.

@en

The antitumor agent imexon act ...... an oxidative stress response.

@nl

type

label

The antitumor agent imexon act ...... an oxidative stress response.

@en

The antitumor agent imexon act ...... an oxidative stress response.

@nl

prefLabel

The antitumor agent imexon act ...... an oxidative stress response.

@en

The antitumor agent imexon act ...... an oxidative stress response.

@nl

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1078-0432.CCR-06-0873

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Clinical Cancer Research

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The antitumor agent imexon act ...... an oxidative stress response.

@en

P2093

Amanda F Baker

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Amy Coon

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Breonna E Tavenner

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Jaime M C Gard

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Robert Dorr

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Terry Landowski

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Tomislov Dragovich

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Wendy R Tate

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P2860

Cloning and expression of a novel human glutaredoxin (Grx2) with mitochondrial and nuclear isoforms

Nrf1 and Nrf2 positively and c-Fos and Fra1 negatively regulate the human antioxidant response element-mediated expression of NAD(P)H:quinone oxidoreductase1 gene

The Nrf2 transcription factor contributes both to the basal expression of glutathione S-transferases in mouse liver and to their induction by the chemopreventive synthetic antioxidants, butylated hydroxyanisole and ethoxyquin

Structure, mechanism and regulation of peroxiredoxins

Identification of Nrf2-regulated genes induced by the chemopreventive agent sulforaphane by oligonucleotide microarray

An Nrf2/small Maf heterodimer mediates the induction of phase II detoxifying enzyme genes through antioxidant response elements

Nrf2-Keap1 defines a physiologically important stress response mechanism

Nrf2 and Nrf1 in association with Jun proteins regulate antioxidant response element-mediated expression and coordinated induction of genes encoding detoxifying enzymes

Antiproliferative and antitumor activity of the 2-cyanoaziridine compound imexon on tumor cell lines and fresh tumor cells in vitro

Nrf3 negatively regulates antioxidant-response element-mediated expression and antioxidant induction of NAD(P)H:quinone oxidoreductase1 gene

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3388-3394

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scholarly article

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10.1158/1078-0432.CCR-06-0873

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11

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