Soluble IL-2RA levels in multiple sclerosis subjects and the effect of soluble IL-2RA on immune responses - Wikidata - wikimedia - TriplyDB

Soluble IL-2RA levels in multiple sclerosis subjects and the effect of soluble IL-2RA on immune responses

Soluble IL-2RA levels in multiple sclerosis subjects and the effect of soluble IL-2RA on immune responses

http://www.wikidata.org/entity/Q37712459

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Translational utility of experimental autoimmune encephalomyelitis: recent developments Daclizumab therapy for multiple sclerosis Therapeutic efficacy of monthly subcutaneous injection of daclizumab in relapsing multiple sclerosis EBI2 augments Tfh cell fate by promoting interaction with IL-2-quenching dendritic cells Genome-wide association analysis of autoantibody positivity in type 1 diabetes cases.An ImmunoChip study of multiple sclerosis risk in African Americans.Influence of membrane CD25 stability on T lymphocyte activity: implications for immunoregulation.Development of a quantitative bead capture assay for soluble IL-7 receptor alpha in human plasma.Fine-mapping resolves Eae23 into two QTLs and implicates ZEB1 as a candidate gene regulating experimental neuroinflammation in rat.BCL6 controls Th9 cell development by repressing Il9 transcription.IL-7 in human health and disease.A case of post-transplant adult T-cell leukemia/lymphoma presenting myelopathy similar to but distinct from human T-cell leukemia virus type I (HTLV- I)-associated myelopathy.Soluble IL-2Rα (sCD25) exacerbates autoimmunity and enhances the development of Th17 responses in mice Beyond regulatory T cells: the potential role for IL-2 to deplete T-follicular helper cells and treat autoimmune diseases.Multiple autoimmune-associated variants confer decreased IL-2R signaling in CD4+ CD25(hi) T cells of type 1 diabetic and multiple sclerosis patients Genetics and the environment converge to dysregulate N-glycosylation in multiple sclerosis.CD4+FOXP3+ T regulatory cells in human autoimmunity: more than a numbers game Hepatocellular carcinoma immunopathogenesis: clinical evidence for global T cell defects and an immunomodulatory role for soluble CD25 (sCD25).Genetic basis of alopecia areata: a roadmap for translational research.Immune derangements in patients with myelofibrosis: the role of Treg, Th17, and sIL2Rα.Soluble IL-2Rα facilitates IL-2-mediated immune responses and predicts reduced survival in follicular B-cell non-Hodgkin lymphoma.A T cell extrinsic mechanism by which IL-2 dampens Th17 differentiation.Associations of Independent IL2RA Gene Variants with Intermediate Uveitis Hypomorphic MGAT5 polymorphisms promote multiple sclerosis cooperatively with MGAT1 and interleukin-2 and 7 receptor variants.IL2RA is associated with persistence of rheumatoid arthritis.Rapamycin/IL-2 combination therapy in patients with type 1 diabetes augments Tregs yet transiently impairs β-cell function.Interleukin-2 receptor-α proximal promoter hypomethylation is associated with multiple sclerosis.The single nucleotide variant rs12722489 determines differential estrogen receptor binding and enhancer properties of an IL2RA intronic region Anticancer immunotherapy by CTLA-4 blockade: obligatory contribution of IL-2 receptors and negative prognostic impact of soluble CD25.Soluble IL7Rα potentiates IL-7 bioactivity and promotes autoimmunity.From markers to molecular mechanisms: type 1 diabetes in the post-GWAS era.Clinical relevance and functional consequences of the TNFRSF1A multiple sclerosis locus.Cell-specific protein phenotypes for the autoimmune locus IL2RA using a genotype-selectable human bioresource.Comparative multiplex analysis of cytokines, chemokines and growth factors in follicular fluid of normoresponder women undergoing ovum donation with gonadotropin-releasing hormone agonist versus gonadotropin-releasing hormone antagonist protocols.Plasma concentrations of soluble IL-2 receptor α (CD25) are increased in type 1 diabetes and associated with reduced C-peptide levels in young patients.Autoimmunity risk alleles in costimulation pathways.From genes to function: the next challenge to understanding multiple sclerosis.Pathogenesis of multiple sclerosis via environmental and genetic dysregulation of N-glycosylation.Multiple sclerosis genetics--is the glass half full, or half empty?Type 1 diabetes: etiology, immunology, and therapeutic strategies.

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Soluble IL-2RA levels in multiple sclerosis subjects and the effect of soluble IL-2RA on immune responses

http://www.wikidata.org/entity/Q37712459

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article científic

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article scientifique

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bilimsel makale

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scientific article published on February 2009

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name

Soluble IL-2RA levels in multi ...... ble IL-2RA on immune responses

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Soluble IL-2RA levels in multi ...... le IL-2RA on immune responses.

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Soluble IL-2RA levels in multi ...... ble IL-2RA on immune responses

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Soluble IL-2RA levels in multi ...... le IL-2RA on immune responses.

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Soluble IL-2RA levels in multi ...... ble IL-2RA on immune responses

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Soluble IL-2RA levels in multi ...... le IL-2RA on immune responses.

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JIMMUNOL.182.3.1541

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Journal of Immunology

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Soluble IL-2RA levels in multi ...... ble IL-2RA on immune responses

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Brian Healy

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Christopher A Severson

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Clare Baecher-Allan

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David E Anderson

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David V Liu

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K Dane Wittrup

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Lisa M Maier

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Philip L De Jager

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P2860

Recommended diagnostic criteria for multiple sclerosis: Guidelines from the international panel on the diagnosis of multiple sclerosis

Risk alleles for multiple sclerosis identified by a genomewide study

Jaks and STATs: biological implications

Axonal pathology in multiple sclerosis: relationship to neurologic disability.

Clinically isolated syndromes suggestive of multiple sclerosis, part 2: non-conventional MRI, recovery processes, and management.

IL2RA genetic heterogeneity in multiple sclerosis and type 1 diabetes susceptibility and soluble interleukin-2 receptor production.

The soluble interleukin 6 receptor: mechanisms of production and implications in disease.

Loss of functional suppression by CD4+CD25+ regulatory T cells in patients with multiple sclerosis.

Foxp3+ CD25+ CD4+ natural regulatory T cells in dominant self-tolerance and autoimmune disease.

In vivo activated T lymphocytes in the peripheral blood and cerebrospinal fluid of patients with multiple sclerosis.

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1541-1547

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scholarly article

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10.4049/JIMMUNOL.182.3.1541

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3

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182

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David A. Hafler

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2009-02-01T00:00:00Z

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23803350

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19155502

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multiple sclerosis

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3992946

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