STAT3 genotypic variation and cellular STAT3 activation and colon leukocyte recruitment in pediatric Crohn disease. - Wikidata - wikimedia - TriplyDB

STAT3 genotypic variation and cellular STAT3 activation and colon leukocyte recruitment in pediatric Crohn disease.

STAT3 genotypic variation and cellular STAT3 activation and colon leukocyte recruitment in pediatric Crohn disease.

http://www.wikidata.org/entity/Q37718730

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Dendritic cells from Crohn's disease patients show aberrant STAT1 and STAT3 signaling Stat3 activation in murine colitis induced by enterotoxigenic Bacteroides fragilis.MicroRNA-124 regulates STAT3 expression and is down-regulated in colon tissues of pediatric patients with ulcerative colitis.Associations between STAT3 rs744166 polymorphisms and susceptibility to ulcerative colitis and Crohn's disease: a meta-analysis Signal transducer and activator of transcription 3: a year in review.Targeting STAT3 in gastric cancer.Immune deficiency vs. immune excess in inflammatory bowel diseases-STAT3 as a rheo-STAT of intestinal homeostasis.Tissue-infiltrating neutrophils represent the main source of IL-23 in the colon of patients with IBD.FNDC4 acts as an anti-inflammatory factor on macrophages and improves colitis in mice.Deletion of intestinal epithelial cell STAT3 promotes T-lymphocyte STAT3 activation and chronic colitis following acute dextran sodium sulfate injury in mice.Paediatric Crohn disease patients with stricturing behaviour exhibit ileal granulocyte-macrophage colony-stimulating factor (GM-CSF) autoantibody production and reduced neutrophil bacterial killing and GM-CSF bioactivity.Mapping hyper-susceptibility to colitis-associated colorectal cancer in FVB/NJ mice.Pilot study of variants of the IL-23R and STAT3 genes reveals no association with Hashimoto's thyroiditis in the Croatian population.

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STAT3 genotypic variation and cellular STAT3 activation and colon leukocyte recruitment in pediatric Crohn disease.

http://www.wikidata.org/entity/Q37718730

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on July 2012

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

STAT3 genotypic variation and ...... nt in pediatric Crohn disease.

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STAT3 genotypic variation and ...... nt in pediatric Crohn disease.

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STAT3 genotypic variation and ...... nt in pediatric Crohn disease.

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STAT3 genotypic variation and ...... nt in pediatric Crohn disease.

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STAT3 genotypic variation and ...... nt in pediatric Crohn disease.

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STAT3 genotypic variation and ...... nt in pediatric Crohn disease.

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Journal of Pediatric Gastroenterology and Nutrition

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STAT3 genotypic variation and ...... nt in pediatric Crohn disease.

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Benjamin R Kuhn

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David Moon

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Erin Bonkowski

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Huan Xu

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Ingrid Jurickova

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Lee A Denson

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Margaret H Collins

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Rebecca Carey

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Shaina Gerad

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Stephen L Guthery

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P2860

Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease

Janus kinases in immune cell signaling

Common variants at five new loci associated with early-onset inflammatory bowel disease

Inhibition of dendritic cell differentiation and accumulation of myeloid-derived suppressor cells in cancer is regulated by S100A9 protein

Structural and thermodynamic characterization of the TYK2 and JAK3 kinase domains in complex with CP-690550 and CMP-6

Characterization of synthetic human granulocyte chemotactic protein 2: usage of chemokine receptors CXCR1 and CXCR2 and in vivo inflammatory properties

IL-6 and Stat3 are required for survival of intestinal epithelial cells and development of colitis-associated cancer

Proteins encoded in genomic regions associated with immune-mediated disease physically interact and suggest underlying biology.

STAT3 controls the neutrophil migratory response to CXCR2 ligands by direct activation of G-CSF-induced CXCR2 expression and via modulation of CXCR2 signal transduction

CD4+ regulatory T cells control TH17 responses in a Stat3-dependent manner.

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32-43

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