Cellular Senescence is a Common Characteristic Shared by Preneoplasic and Osteo-Arthritic Tissue. - Wikidata - wikimedia - TriplyDB

Cellular Senescence is a Common Characteristic Shared by Preneoplasic and Osteo-Arthritic Tissue.

Cellular Senescence is a Common Characteristic Shared by Preneoplasic and Osteo-Arthritic Tissue.

http://www.wikidata.org/entity/Q37721188

about

The inhibitory effect of salmon calcitonin on tri-iodothyronine induction of early hypertrophy in articular cartilage Chondrogenic differentiation of mesenchymal stem cells: challenges and unfulfilled expectations.[Cellular senescence and the myth of Janus].

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P2860

Cellular Senescence is a Common Characteristic Shared by Preneoplasic and Osteo-Arthritic Tissue.

http://www.wikidata.org/entity/Q37721188

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 11 February 2010

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Cellular Senescence is a Commo ...... ic and Osteo-Arthritic Tissue.

@en

Cellular Senescence is a Commo ...... ic and Osteo-Arthritic Tissue.

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type

label

Cellular Senescence is a Commo ...... ic and Osteo-Arthritic Tissue.

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Cellular Senescence is a Commo ...... ic and Osteo-Arthritic Tissue.

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Cellular Senescence is a Commo ...... ic and Osteo-Arthritic Tissue.

@en

Cellular Senescence is a Commo ...... ic and Osteo-Arthritic Tissue.

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1874312901004010010

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The open rheumatology journal

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Cellular Senescence is a Commo ...... ic and Osteo-Arthritic Tissue.

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Christian Jorgensen

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Danièle Noël

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Didier Philipot

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Jean-Marc Brondello

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P2860

Potential involvement of oxidative stress in cartilage senescence and development of osteoarthritis: oxidative stress induces chondrocyte telomere instability and downregulation of chondrocyte function.

Oncogene-induced senescence relayed by an interleukin-dependent inflammatory network.

Chemokine signaling via the CXCR2 receptor reinforces senescence

Integrative microRNA and proteomic approaches identify novel osteoarthritis genes and their collaborative metabolic and inflammatory networks

Senescence-associated secretory phenotypes reveal cell-nonautonomous functions of oncogenic RAS and the p53 tumor suppressor

Cellular senescence: when bad things happen to good cells

Oncogene-induced senescence is a DNA damage response triggered by DNA hyper-replication

Unexpected pieces to the senescence puzzle

Osteoarthritis: new insights. Part 1: the disease and its risk factors.

Composition and structure of articular cartilage: a template for tissue repair.

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