The mitochondria permeability transition pore complex in the brain with interacting proteins - promising targets for protection in neurodegenerative diseases.
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Brain Lipotoxicity of Phytanic Acid and Very Long-chain Fatty Acids. Harmful Cellular/Mitochondrial Activities in Refsum Disease and X-Linked AdrenoleukodystrophyWhy does brain metabolism not favor burning of fatty acids to provide energy? Reflections on disadvantages of the use of free fatty acids as fuel for brainProtective effects and mechanisms of sirtuins in the nervous systemRegulation of the inner membrane mitochondrial permeability transition by the outer membrane translocator protein (peripheral benzodiazepine receptor)Carbenoxolone induces permeability transition pore opening in rat mitochondria via the translocator protein TSPO and connexin43.Evolving understanding of translocator protein 18 kDa (TSPO)Purines: forgotten mediators in traumatic brain injury.Combined effect of G3139 and TSPO ligands on Ca(2+)-induced permeability transition in rat brain mitochondriaRenal 2',3'-Cyclic Nucleotide 3'-Phosphodiesterase Is an Important Determinant of AKI Severity after Ischemia-Reperfusion.Multi-Target-Directed Ligands and other Therapeutic Strategies in the Search of a Real Solution for Alzheimer's Disease.Calcium dysregulation and homeostasis of neural calcium in the molecular mechanisms of neurodegenerative diseases provide multiple targets for neuroprotection.Mitochondrial dysfunction--a pharmacological target in Alzheimer's disease.Neurovascular pathophysiology in cerebral ischemia, dementia and the ageing brain - current trends in basic, translational and clinical research.Mitochondrial channels: ion fluxes and more.zRICH, a protein induced during optic nerve regeneration in zebrafish, promotes neuritogenesis and interacts with tubulin.Mitochondrial Ca(2+) Processing by a Unit of Mitochondrial Ca(2+) Uniporter and Na(+)/Ca(2+) Exchanger Supports the Neuronal Ca(2+) Influx via Activated Glutamate Receptors.Biochemical Evidence for a Putative Inositol 1,3,4,5-Tetrakisphosphate Receptor in the Olfactory System of Atlantic Salmon (Salmo salar)The 2009 meeting of the study group 'Molecular Neurobiology' of the German Society for Biochemistry and Molecular Biology.Mitoprotective effect of Centella asiatica against aluminum-induced neurotoxicity in rats: possible relevance to its anti-oxidant and anti-apoptosis mechanism.Discovery and Roles of 2',3'-cAMP in Biological Systems.Anti-Oxidant and Anti-Inflammatory Activity of Ketogenic Diet: New Perspectives for Neuroprotection in Alzheimer's Disease.
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P2860
The mitochondria permeability transition pore complex in the brain with interacting proteins - promising targets for protection in neurodegenerative diseases.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on June 2010
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
The mitochondria permeability ...... in neurodegenerative diseases.
@en
The mitochondria permeability ...... in neurodegenerative diseases.
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type
label
The mitochondria permeability ...... in neurodegenerative diseases.
@en
The mitochondria permeability ...... in neurodegenerative diseases.
@nl
prefLabel
The mitochondria permeability ...... in neurodegenerative diseases.
@en
The mitochondria permeability ...... in neurodegenerative diseases.
@nl
P2093
P2860
P356
P1433
P1476
The mitochondria permeability ...... in neurodegenerative diseases.
@en
P2093
Georg Reiser
Rolf Stricker
Tamara Azarashvili
P2860
P304
P356
10.1515/BC.2010.070
P577
2010-06-01T00:00:00Z