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Going up in flames: necrotic cell injury and inflammatory diseases.

Going up in flames: necrotic cell injury and inflammatory diseases.

http://www.wikidata.org/entity/Q37763609

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Programmed necrosis in acute kidney injury Viral infection and the evolution of caspase 8-regulated apoptotic and necrotic death pathways Necrotic Cells Actively Attract Phagocytes through the Collaborative Action of Two Distinct PS-Exposure Mechanisms The role of necroptosis in neurosurgical diseases RIP1-dependent and independent effects of necrostatin-1 in necrosis and T cell activation Clostridium perfringens beta-toxin induces necrostatin-inhibitable, calpain-dependent necrosis in primary porcine endothelial cells Retinal ganglion cell (RGC) programmed necrosis contributes to ischemia-reperfusion-induced retinal damage.Pro-apoptotic versus anti-apoptotic properties of dietary resveratrol on tumoral and normal cardiac cells.Necroptosis Genetic control of necrosis - another type of programmed cell death.Tumor necrosis factor-alpha mediates activation of NF-κB and JNK signaling cascades in retinal ganglion cells and astrocytes in opposite ways.A nonredundant role for plasmacytoid dendritic cells in host defense against the human fungal pathogen Aspergillus fumigatus.CYLD deubiquitinates RIP1 in the TNFα-induced necrosome to facilitate kinase activation and programmed necrosis.Helicobacter pylori VacA induces programmed necrosis in gastric epithelial cells.Chronic Treatment with a Water-Soluble Extract from the Culture Medium of Ganoderma lucidum Mycelia Prevents Apoptosis and Necroptosis in Hypoxia/Ischemia-Induced Injury of Type 2 Diabetic Mouse Brain Recognition of Aspergillus fumigatus hyphae by human plasmacytoid dendritic cells is mediated by dectin-2 and results in formation of extracellular traps.Increased HMGB1 and cleaved caspase-3 stimulate the proliferation of tumor cells and are correlated with the poor prognosis in colorectal cancer.Inflammation, endoplasmic reticulum stress, autophagy, and the monocyte chemoattractant protein-1/CCR2 pathway.Dichotomy between RIP1- and RIP3-mediated necroptosis in tumor necrosis factor-α-induced shock.Continuum modeling of a neuronal cell under blast loading The RIP1-kinase inhibitor necrostatin-1 prevents osmotic nephrosis and contrast-induced AKI in mice.Are reactive oxygen species always detrimental to pathogens?Programmed cell death in the plant immune system Death and inflammation following somatic cell transplantation.Inhibitor of apoptosis proteins: fascinating biology leads to attractive tumor therapeutic targets.Macrophages and recently identified forms of cell death.Viral cell death inhibitor MC159 enhances innate immunity against vaccinia virus infection.Preferential induction of apoptotic cell death in melanoma cells as compared with normal keratinocytes using a non-thermal plasma torch.BCL2 suppresses PARP1 function and nonapoptotic cell death.Necroptosis in neurodegenerative diseases: a potential therapeutic target.How are necrotic cells recognized by their predators?Monitoring the clearance of apoptotic and necrotic cells in the nematode Caenorhabditis elegans.Active leukocyte detachment and apoptosis/necrosis on PEG hydrogels and the implication in the host inflammatory response Detection of necrosis by release of lactate dehydrogenase activity.Heme induces programmed necrosis on macrophages through autocrine TNF and ROS production.Cell Death Pathways: a Novel Therapeutic Approach for Neuroscientists.Necrostatin-1 inhibits Hmgb1-IL-23/IL-17 pathway and attenuates cardiac ischemia reperfusion injury.RIPK3-mediated necroptosis promotes donor kidney inflammatory injury and reduces allograft survival.RIPK1-RIPK3-MLKL-Associated Necroptosis Drives Killing in Neutrophils

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P2860

Going up in flames: necrotic cell injury and inflammatory diseases.

http://www.wikidata.org/entity/Q37763609

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 08 June 2010

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

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name

Going up in flames: necrotic cell injury and inflammatory diseases.

@en

Going up in flames: necrotic cell injury and inflammatory diseases.

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type

label

Going up in flames: necrotic cell injury and inflammatory diseases.

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Going up in flames: necrotic cell injury and inflammatory diseases.

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prefLabel

Going up in flames: necrotic cell injury and inflammatory diseases.

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Going up in flames: necrotic cell injury and inflammatory diseases.

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Cellular and Molecular Life Sciences

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Going up in flames: necrotic cell injury and inflammatory diseases.

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Francis Ka-Ming Chan

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Sreerupa Challa

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P2860

Programmed cellular necrosis mediated by the pore-forming alpha-toxin from Clostridium septicum

Recruitment of the linear ubiquitin chain assembly complex stabilizes the TNF-R1 signaling complex and is required for TNF-mediated gene induction

Cleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosis

Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule

De-ubiquitination and ubiquitin ligase domains of A20 downregulate NF-kappaB signalling

Inhibition of ADP/ATP exchange in receptor-interacting protein-mediated necrosis.

RIP: a novel protein containing a death domain that interacts with Fas/APO-1 (CD95) in yeast and causes cell death

DAI/ZBP1 recruits RIP1 and RIP3 through RIP homotypic interaction motifs to activate NF-kappaB

TNF-alpha induces two distinct caspase-8 activation pathways

Involvement of linear polyubiquitylation of NEMO in NF-kappaB activation

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s00018-010-0413-8

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3241-3253

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scholarly article

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10.1007/S00018-010-0413-8

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19

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67

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2010-06-08T00:00:00Z

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20532807

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