Cognitive deficits in experimental autoimmune encephalomyelitis: neuroinflammation and synaptic degeneration.
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Impaired sprouting and axonal atrophy in cerebellar climbing fibres following in vivo silencing of the growth-associated protein GAP-43Pediatric Autoimmune Disorders Associated with Streptococcal Infections and Tourette's Syndrome in Preclinical Studies.The "quad-partite" synapse: microglia-synapse interactions in the developing and mature CNSNeuroendocrine immunoregulation in multiple sclerosisAltered excitatory-inhibitory balance within somatosensory cortex is associated with enhanced plasticity and pain sensitivity in a mouse model of multiple sclerosis.Balancing the immune response in the brain: IL-10 and its regulationImpact of natalizumab on cognitive performances and fatigue in relapsing multiple sclerosis: a prospective, open-label, two years observational studyInflammation subverts hippocampal synaptic plasticity in experimental multiple sclerosis.Histamine H(3) receptor integrates peripheral inflammatory signals in the neurogenic control of immune responses and autoimmune disease susceptibilityAssociation between a genetic variant of type-1 cannabinoid receptor and inflammatory neurodegeneration in multiple sclerosisMechanism of experimental autoimmune encephalomyelitis in Lewis rats: recent insights from macrophages.LPS-induced Murine Neuroinflammation Model: Main Features and Suitability for Pre-clinical Assessment of NutraceuticalsSaturable leptin transport across the BBB persists in EAE miceActivation of Wnt signaling promotes hippocampal neurogenesis in experimental autoimmune encephalomyelitis.Synaptic plasticity in multiple sclerosis and in experimental autoimmune encephalomyelitis.Selective vulnerability of synaptic signaling and metabolism to nitrosative stressOlfactory dysfunction: its early temporal relationship and neural correlates in the pathogenesis of Alzheimer's disease.Natalizumab discontinuation is associated with a rebound of cognitive impairment in multiple sclerosis patients.Glatiramer acetate guards against rapid memory decline during relapsing-remitting experimental autoimmune encephalomyelitis.Targeting synaptic pathology in multiple sclerosis: fingolimod to the rescue?The neuroinflammatory response in humans after traumatic brain injury.Interleukin-1β promotes long-term potentiation in patients with multiple sclerosis.Glutamate release machinery is altered in the frontal cortex of rats with experimental autoimmune encephalomyelitis.18F-VC701-PET and MRI in the in vivo neuroinflammation assessment of a mouse model of multiple sclerosis.Retrograde interferon-gamma signaling induces major histocompatibility class I expression in human-induced pluripotent stem cell-derived neurons.
P2860
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P2860
Cognitive deficits in experimental autoimmune encephalomyelitis: neuroinflammation and synaptic degeneration.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh
2010年學術文章
@zh-hant
name
Cognitive deficits in experime ...... ion and synaptic degeneration.
@en
Cognitive deficits in experime ...... ion and synaptic degeneration.
@nl
type
label
Cognitive deficits in experime ...... ion and synaptic degeneration.
@en
Cognitive deficits in experime ...... ion and synaptic degeneration.
@nl
prefLabel
Cognitive deficits in experime ...... ion and synaptic degeneration.
@en
Cognitive deficits in experime ...... ion and synaptic degeneration.
@nl
P1476
Cognitive deficits in experime ...... ion and synaptic degeneration.
@en
P2093
G Mandolesi
G Musumeci
P2888
P304
P356
10.1007/S10072-010-0369-3
P433
P577
2010-11-01T00:00:00Z