Interleukin-1β promotes long-term potentiation in patients with multiple sclerosis. - Wikidata - wikimedia - TriplyDB

Interleukin-1β promotes long-term potentiation in patients with multiple sclerosis.

Interleukin-1β promotes long-term potentiation in patients with multiple sclerosis.

http://www.wikidata.org/entity/Q46899417

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Neural Plasticity in Multiple Sclerosis: The Functional and Molecular Background Investigating perturbed pathway modules from gene expression data via structural equation models.Cortical excitability changes over time in progressive multiple sclerosis.Neuroimmune mechanisms of alcohol and drug addiction.Interaction between interleukin-1β and type-1 cannabinoid receptor is involved in anxiety-like behavior in experimental autoimmune encephalomyelitis Synaptic plasticity in multiple sclerosis and in experimental autoimmune encephalomyelitis.IL-1β dependent cerebellar synaptopathy in a mouse mode of multiple sclerosis.Inflammatory demyelination alters subcortical visual circuits.Synaptopathy connects inflammation and neurodegeneration in multiple sclerosis.Can we define a rehabilitation strategy for cognitive impairment in progressive multiple sclerosis? A critical appraisal.Phospholipase D1 expression analysis in relapsing-remitting multiple sclerosis patients.Synaptoimmunology - roles in health and disease.Siponimod (BAF312) prevents synaptic neurodegeneration in experimental multiple sclerosis Palmitoylethanolamide Reduces Neuropsychiatric Behaviors by Restoring Cortical Electrophysiological Activity in a Mouse Model of Mild Traumatic Brain Injury.RANTES correlates with inflammatory activity and synaptic excitability in multiple sclerosis.Amyloid-β Homeostasis Bridges Inflammation, Synaptic Plasticity Deficits and Cognitive Dysfunction in Multiple Sclerosis.Platelet-derived growth factor predicts prolonged relapse-free period in multiple sclerosis.Tumor Necrosis Factor and Interleukin-1β Modulate Synaptic Plasticity during Neuroinflammation.Neuroimmune Activation Drives Multiple Brain States Depression following a traumatic brain injury: uncovering cytokine dysregulation as a pathogenic mechanism Interplay Between Age and Neuroinflammation in Multiple Sclerosis: Effects on Motor and Cognitive Functions TRPV4 Inhibition Improved Myelination and Reduced Glia Reactivity and Inflammation in a Cuprizone-Induced Mouse Model of Demyelination

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Interleukin-1β promotes long-term potentiation in patients with multiple sclerosis.

http://www.wikidata.org/entity/Q46899417

description

2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年學術文章

@yue

2013年學術文章

@zh-hant

name

Interleukin-1β promotes long-term potentiation in patients with multiple sclerosis.

@en

Interleukin-1β promotes long-term potentiation in patients with multiple sclerosis.

@nl

type

label

Interleukin-1β promotes long-term potentiation in patients with multiple sclerosis.

@en

Interleukin-1β promotes long-term potentiation in patients with multiple sclerosis.

@nl

prefLabel

Interleukin-1β promotes long-term potentiation in patients with multiple sclerosis.

@en

Interleukin-1β promotes long-term potentiation in patients with multiple sclerosis.

@nl

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NeuroMolecular Medicine

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Interleukin-1β promotes long-term potentiation in patients with multiple sclerosis

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Alessandra Bergami

http://www.w3.org/2001/XMLSchema#string

Carolina G Nicoletti

http://www.w3.org/2001/XMLSchema#string

Fabio Buttari

http://www.w3.org/2001/XMLSchema#string

Ferdinando Nicoletti

http://www.w3.org/2001/XMLSchema#string

Gianvito Martino

http://www.w3.org/2001/XMLSchema#string

Hajime Kusayanagi

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P2860

Astrogliosis in CNS pathologies: is there a role for microglia?

Diagnostic criteria for multiple sclerosis: 2010 revisions to the McDonald criteria

GABA(A) receptor-mediated acceleration of aging-associated memory decline in APP/PS1 mice and its pharmacological treatment by picrotoxin

Inflammation-mediated memory dysfunction and effects of a ketogenic diet in a murine model of multiple sclerosis

Adaptive functional changes in the cerebral cortex of patients with nondisabling multiple sclerosis correlate with the extent of brain structural damage.

Impact of acute inflammation on spinal motoneuron synaptic plasticity following ventral root avulsion.

In vivo imaging of lymphocytes in the CNS reveals different behaviour of naïve T cells in health and autoimmunity.

Experience-dependent modification of synaptic plasticity in visual cortex.

Excitability decreasing central motor plasticity is retained in multiple sclerosis patients.

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s12017-013-8249-7

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1

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16

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Georgia Mandolesi

Nicola B. Mercuri

Nicola Berretta

Antonietta Gentile

Alessandra Musella

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2013-07-28T00:00:00Z

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multiple sclerosis