Cellular metabolic stress: considering how cells respond to nutrient excess.
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Targeting mitochondrial reactive oxygen species as novel therapy for inflammatory diseases and cancersErk regulation of pyruvate dehydrogenase flux through PDK4 modulates cell proliferationThe crucial impact of lysosomes in aging and longevityLeishmania carbon metabolism in the macrophage phagolysosome- feast or famine?Diabetes and cancer: two diseases with obesity as a common risk factorMolecular mechanisms of superoxide production by complex III: a bacterial versus human mitochondrial comparative case studyRole of nutrient-sensing signals in the pathogenesis of diabetic nephropathymTOR-dependent cell survival mechanismsThe Role of Bioactive Dietary Components in Modulating miRNA Expression in Colorectal CancerInflammasomes and metabolic disorders: old genes in modern diseasesPro-Oxidant Role of Silibinin in DMBA/TPA Induced Skin Cancer: 1H NMR Metabolomic and Biochemical StudyRegulation of the mTOR complex 1 pathway by nutrients, growth factors, and stressPlacental Origins of Chronic Disease.Alterations in c-Myc phenotypes resulting from dynamin-related protein 1 (Drp1)-mediated mitochondrial fission.Targeting the mitochondria activates two independent cell death pathways in ovarian cancer stem cellsEffect of the cancer specific shorter form of human 6-phosphofructo-1-kinase on the metabolism of the yeast Saccharomyces cerevisiae.Regulation of CHK1 by mTOR contributes to the evasion of DNA damage barrier of cancer cellsAssociation between hyperinsulinemia and increased risk of cancer death in nonobese and obese people: A population-based observational studyGene expression in teratogenic exposures: a new approach to understanding individual risk.Epigenetics: Linking Nutrition to Molecular Mechanisms in Aging.C. elegans SUP-46, an HNRNPM family RNA-binding protein that prevents paternally-mediated epigenetic sterility.The multi-level action of fatty acids on adiponectin production by fat cellsOpen chromatin profiling in mice livers reveals unique chromatin variations induced by high fat diet.The aging stress response.REGγ is associated with multiple oncogenic pathways in human cancers.Dynamic modelling of oestrogen signalling and cell fate in breast cancer cellsInhibition of pyruvate kinase M2 by reactive oxygen species contributes to cellular antioxidant responsesA two-way street: reciprocal regulation of metabolism and signalling.Proton-assisted amino acid transporter PAT1 complexes with Rag GTPases and activates TORC1 on late endosomal and lysosomal membranes.Association of polymorphisms in oxidative stress genes with clinical outcomes for bladder cancer treated with Bacillus Calmette-Guérin.The hexosamine biosynthetic pathway couples growth factor-induced glutamine uptake to glucose metabolism.LPS from P. gingivalis and hypoxia increases oxidative stress in periodontal ligament fibroblasts and contributes to periodontitisMetabolic imaging: a link between lactate dehydrogenase A, lactate, and tumor phenotype.The Warburg Effect: How Does it Benefit Cancer Cells?Metabolic responses of primary and transformed cells to intracellular Listeria monocytogenes.Epigenomic regulation of host-microbiota interactions.Hypoxia. 2. Hypoxia regulates cellular metabolismC-Terminal Binding Protein: A Molecular Link between Metabolic Imbalance and Epigenetic Regulation in Breast Cancer.Synthetic lethality of combined glutaminase and Hsp90 inhibition in mTORC1-driven tumor cellsHDAC1 and HDAC2 restrain the intestinal inflammatory response by regulating intestinal epithelial cell differentiation.
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Cellular metabolic stress: considering how cells respond to nutrient excess.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on October 2010
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vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Cellular metabolic stress: considering how cells respond to nutrient excess.
@en
Cellular metabolic stress: considering how cells respond to nutrient excess.
@nl
type
label
Cellular metabolic stress: considering how cells respond to nutrient excess.
@en
Cellular metabolic stress: considering how cells respond to nutrient excess.
@nl
prefLabel
Cellular metabolic stress: considering how cells respond to nutrient excess.
@en
Cellular metabolic stress: considering how cells respond to nutrient excess.
@nl
P2860
P1433
P1476
Cellular metabolic stress: considering how cells respond to nutrient excess
@en
P2093
Craig B Thompson
P2860
P304
P356
10.1016/J.MOLCEL.2010.10.004
P577
2010-10-01T00:00:00Z