Senescence-associated intrinsic mechanisms of osteoblast dysfunctions.
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Skeletal stem cells and their contribution to skeletal fragility: senescence and rejuvenationThe immunomodulatory and neuroprotective effects of mesenchymal stem cells (MSCs) in experimental autoimmune encephalomyelitis (EAE): a model of multiple sclerosis (MS)Skeletal stem cellsChanges in primary lymphoid organs with agingRoles of FGF signaling in stem cell self-renewal, senescence and agingMouse models of telomere dysfunction phenocopy skeletal changes found in human age-related osteoporosis.Aging diminishes lamellar and woven bone formation induced by tibial compression in adult C57BL/6Human immunodeficiency virus envelope protein Gp120 induces proliferation but not apoptosis in osteoblasts at physiologic concentrations.Cellular senescence: a link between cancer and age-related degenerative disease?Effect of GH/IGF-1 on Bone Metabolism and Osteoporsosis.Protein kinase Cα (PKCα) regulates bone architecture and osteoblast activity.Increased marrow adiposity in premenopausal women with idiopathic osteoporosisAge-related impairment of bones' adaptive response to loading in mice is associated with sex-related deficiencies in osteoblasts but no change in osteocytes.Loss of the osteogenic differentiation potential during senescence is limited to bone progenitor cells and is dependent on p53.Euodia sutchuenensis Dode extract stimulates osteoblast differentiation via Wnt/β-catenin pathway activationThe role of microRNAs in cellular senescence and age-related conditions of cartilage and bone.Age-related decline in osteoblastogenesis and 1α-hydroxylase/CYP27B1 in human mesenchymal stem cells: stimulation by parathyroid hormone.Pharmacological Inhibition of Protein Kinase G1 Enhances Bone Formation by Human Skeletal Stem Cells Through Activation of RhoA-Akt Signaling.p47phox-Nox2-dependent ROS Signaling Inhibits Early Bone Development in Mice but Protects against Skeletal Aging.Melatonin reverses H2 O2 -induced premature senescence in mesenchymal stem cells via the SIRT1-dependent pathway.Impairment of osteoblast differentiation due to proliferation-independent telomere dysfunction in mouse models of accelerated agingCombined activation of the energy and cellular-defense pathways may explain the potent anti-senescence activity of methylene blue.Telomerase activity promotes osteoblast differentiation by modulating IGF-signaling pathwayVesicular Galectin-3 levels decrease with donor age and contribute to the reduced osteo-inductive potential of human plasma derived extracellular vesicles.Long-term functional engraftment of mesenchymal progenitor cells in a mouse model of accelerated agingAging alters bone-fat reciprocity by shifting in vivo mesenchymal precursor cell fate towards an adipogenic lineage.Tanshinol Rescues the Impaired Bone Formation Elicited by Glucocorticoid Involved in KLF15 Pathway.Suppression of EZH2 Prevents the Shift of Osteoporotic MSC Fate to Adipocyte and Enhances Bone Formation During Osteoporosisp38α MAPK Regulates Lineage Commitment and OPG Synthesis of Bone Marrow Stromal Cells to Prevent Bone Loss under Physiological and Pathological Conditions.Redundant miR-3077-5p and miR-705 mediate the shift of mesenchymal stem cell lineage commitment to adipocyte in osteoporosis bone marrowAging mechanisms in bone.DNA damage drives accelerated bone aging via an NF-κB-dependent mechanism.Bone and skeletal muscle: neighbors with close tiesBone marrow fat composition as a novel imaging biomarker in postmenopausal women with prevalent fragility fractures.In vitro osteogenic capacity of bone marrow MSCs from postmenopausal women reflect the osseointegration of their cementless hip stems.Pathogenesis of age-related bone loss in humansSATB2-Nanog axis links age-related intrinsic changes of mesenchymal stem cells from craniofacial bone.Loss of Rictor with aging in osteoblasts promotes age-related bone loss.Tanshinol attenuates the deleterious effects of oxidative stress on osteoblastic differentiation via Wnt/FoxO3a signaling.From cellular senescence to age-associated diseases: the miRNA connection.
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P2860
Senescence-associated intrinsic mechanisms of osteoblast dysfunctions.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 18 February 2011
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Senescence-associated intrinsic mechanisms of osteoblast dysfunctions.
@en
Senescence-associated intrinsic mechanisms of osteoblast dysfunctions.
@nl
type
label
Senescence-associated intrinsic mechanisms of osteoblast dysfunctions.
@en
Senescence-associated intrinsic mechanisms of osteoblast dysfunctions.
@nl
prefLabel
Senescence-associated intrinsic mechanisms of osteoblast dysfunctions.
@en
Senescence-associated intrinsic mechanisms of osteoblast dysfunctions.
@nl
P2860
P1433
P1476
Senescence-associated intrinsic mechanisms of osteoblast dysfunctions
@en
P2093
Pierre J Marie
P2860
P304
P356
10.1111/J.1474-9726.2011.00669.X
P577
2011-02-18T00:00:00Z