GABA and central neuropathic pain following spinal cord injury
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Molecular targeting of NOX4 for neuropathic pain after traumatic injury of the spinal cord.Food-Derived Natural Compounds for Pain Relief in Neuropathic PainFatty acid amide hydrolase (FAAH) inhibitors exert pharmacological effects, but lack antinociceptive efficacy in rats with neuropathic spinal cord injury pain.Loss of central inhibition: implications for behavioral hypersensitivity after contusive spinal cord injury in rats.Chronic at-level thermal hyperalgesia following rat cervical contusion spinal cord injury is accompanied by neuronal and astrocyte activation and loss of the astrocyte glutamate transporter, GLT1, in superficial dorsal horn.The current state-of-the-art of spinal cord imaging: applications.Remodeling the Dendritic Spines in the Hindlimb Representation of the Sensory Cortex after Spinal Cord Hemisection in MiceTherapeutic restoration of spinal inhibition via druggable enhancement of potassium-chloride cotransporter KCC2-mediated chloride extrusion in peripheral neuropathic painCombinations of intrathecal gamma-amino-butyrate receptor agonists and N-methyl-d-aspartate receptor antagonists in rats with neuropathic spinal cord injury pain.Gabapentin inhibits γ-amino butyric acid release in the locus coeruleus but not in the spinal dorsal horn after peripheral nerve injury in ratsRodent estrous cycle response to incomplete spinal cord injury, surgical interventions, and locomotor training.Impact of behavioral control on the processing of nociceptive stimulation.Ondansetron reverses antihypersensitivity from clonidine in rats after peripheral nerve injury: role of γ-aminobutyric acid in α2-adrenoceptor and 5-HT3 serotonin receptor analgesia.GLT1 overexpression reverses established neuropathic pain-related behavior and attenuates chronic dorsal horn neuron activation following cervical spinal cord injury.Metabolite concentrations in the anterior cingulate cortex predict high neuropathic pain impact after spinal cord injury.Recombinant neural progenitor transplants in the spinal dorsal horn alleviate chronic central neuropathic pain.The injured and regenerating nervous system: immunoglobulin superfamily members as key players.Spatial and temporal activation of spinal glial cells: role of gliopathy in central neuropathic pain following spinal cord injury in rats.Spinal cord injury pain: mechanisms and management.Opioid administration following spinal cord injury: implications for pain and locomotor recovery.Neuropathic pain: role of inflammation, immune response, and ion channel activity in central injury mechanisms.Spinal cord injury induced neuropathic pain: Molecular targets and therapeutic approaches.Gene expression profiles reveal key pathways and genes associated with neuropathic pain in patients with spinal cord injury.Reactive oxygen species affect spinal cell type-specific synaptic plasticity in a model of neuropathic pain.Molecular and pharmacological evidence for a facilitatory functional role of pre-synaptic GLUK2/3 kainate receptors on GABA release in rat trigeminal caudal nucleus.In vivo microdialysis of glutamate in ventroposterolateral nucleus of thalamus following electrolytic lesion of spinothalamic tract in rats.Purinergic Modulation of Spinal Neuroglial Maladaptive Plasticity Following Peripheral Nerve Injury.K+-Cl- co-transporter 2 (KCC2) - a membrane trafficking perspective.Impaired sensitivity to pain stimuli in plasma membrane calcium ATPase 2 (PMCA2) heterozygous mice: a possible modality- and sex-specific role for PMCA2 in nociception.Multidimensional clinical pain phenotypes after spinal cord injury.When Pain Hurts: Nociceptive Stimulation Induces a State of Maladaptive Plasticity and Impairs Recovery after Spinal Cord Injury.Cognitive assessment of pycnogenol therapy following traumatic brain injury.Estradiol attenuates spinal cord injury-related central pain by decreasing glutamate levels in thalamic VPL nucleus in male rats.Acute spinal cord injury (SCI) transforms how GABA affects nociceptive sensitization.Metaplasticity within the spinal cord: Evidence brain-derived neurotrophic factor (BDNF), tumor necrosis factor (TNF), and alterations in GABA function (ionic plasticity) modulate pain and the capacity to learn.Identification of key genes and pathways involved in response to pain in goat and sheep by transcriptome sequencing
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GABA and central neuropathic pain following spinal cord injury
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 07 January 2011
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
GABA and central neuropathic pain following spinal cord injury
@en
GABA and central neuropathic pain following spinal cord injury.
@nl
type
label
GABA and central neuropathic pain following spinal cord injury
@en
GABA and central neuropathic pain following spinal cord injury.
@nl
prefLabel
GABA and central neuropathic pain following spinal cord injury
@en
GABA and central neuropathic pain following spinal cord injury.
@nl
P2860
P1433
P1476
GABA and central neuropathic pain following spinal cord injury
@en
P2093
Claire E Hulsebosch
Young S Gwak
P2860
P304
P356
10.1016/J.NEUROPHARM.2010.12.030
P577
2011-01-07T00:00:00Z