Macrophages and control of granulomatous inflammation in tuberculosis.
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CXCR5⁺ T helper cells mediate protective immunity against tuberculosisDoes tuberculosis threaten our ageing populations?Th1 and Th17 Cells in Tuberculosis: Protection, Pathology, and BiomarkersImmune vulnerability of infants to tuberculosisStriking the Right Balance Determines TB or Not TBImmunometabolism in TuberculosisComputational and Empirical Studies Predict Mycobacterium tuberculosis-Specific T Cells as a Biomarker for Infection Outcome.Variability in tuberculosis granuloma T cell responses exists, but a balance of pro- and anti-inflammatory cytokines is associated with sterilizationInterferon regulatory factor 8-deficiency determines massive neutrophil recruitment but T cell defect in fast growing granulomas during tuberculosisFirst human model of in vitro Candida albicans persistence within granuloma for the reliable study of host-fungi interactionsBacterial genomic epidemiology, from local outbreak characterization to species-history reconstructionHypoxia inducible factor signaling modulates susceptibility to mycobacterial infection via a nitric oxide dependent mechanismMycobacteria counteract a TLR-mediated nitrosative defense mechanism in a zebrafish infection modelMacrophage polarization drives granuloma outcome during Mycobacterium tuberculosis infectionLow induction of proinflammatory cytokines parallels evolutionary success of modern strains within the Mycobacterium tuberculosis Beijing genotype.Transcript levels of major interleukins in relation to the clinicopathological profile of patients with tuberculous intervertebral discs and healthy controls.Cell-Type Specific Determinants of NRAMP1 Expression in Professional PhagocytesThe role of dendritic cells in driving genital tract inflammation and HIV transmission risk: are there opportunities to intervene?Neutrophils in Tuberculosis: Heterogeneity Shapes the Way?The introduction of mesenchymal stromal cells induces different immunological responses in the lungs of healthy and M. tuberculosis infected mice.The DosS-DosT/DosR Mycobacterial Sensor System.Matrix metalloproteinase proteolysis of the mycobacterial HSP65 protein as a potential source of immunogenic peptides in human tuberculosis.Genome-wide transcriptional profiling of peripheral blood leukocytes from cattle infected with Mycobacterium bovis reveals suppression of host immune genes.Macrophage arginase-1 controls bacterial growth and pathology in hypoxic tuberculosis granulomas.Polymorphisms in the interleukin 18 receptor 1 gene and tuberculosis susceptibility among Chinese.Induction of CCL8/MCP-2 by mycobacteria through the activation of TLR2/PI3K/Akt signaling pathway.Bone marrow mesenchymal stem cells provide an antibiotic-protective niche for persistent viable Mycobacterium tuberculosis that survive antibiotic treatmentMulti-scale modeling predicts a balance of tumor necrosis factor-α and interleukin-10 controls the granuloma environment during Mycobacterium tuberculosis infection.Engrafted human cells generate adaptive immune responses to Mycobacterium bovis BCG infection in humanized mice.Tuberculosis and the risk of infection with other intracellular bacteria: a population-based study.Characterization of host and microbial determinants in individuals with latent tuberculosis infection using a human granuloma model.Immunology studies in non-human primate models of tuberculosisTrans-species communication in the Mycobacterium tuberculosis-infected macrophageCytokine and lipid mediator networks in tuberculosisToll-like receptor 2-dependent extracellular signal-regulated kinase signaling in Mycobacterium tuberculosis-infected macrophages drives anti-inflammatory responses and inhibits Th1 polarization of responding T cellsPPE38 modulates the innate immune response and is required for Mycobacterium marinum virulence.Modeling Granulomas in Response to Infection in the LungJanus-faced liposomes enhance antimicrobial innate immune response in Mycobacterium tuberculosis infectionNF-κB Signaling Dynamics Play a Key Role in Infection Control in Tuberculosis.The tuberculous granuloma: an unsuccessful host defence mechanism providing a safety shelter for the bacteria?
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Macrophages and control of granulomatous inflammation in tuberculosis.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 23 March 2011
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Macrophages and control of granulomatous inflammation in tuberculosis.
@en
Macrophages and control of granulomatous inflammation in tuberculosis.
@nl
type
label
Macrophages and control of granulomatous inflammation in tuberculosis.
@en
Macrophages and control of granulomatous inflammation in tuberculosis.
@nl
prefLabel
Macrophages and control of granulomatous inflammation in tuberculosis.
@en
Macrophages and control of granulomatous inflammation in tuberculosis.
@nl
P2093
P2860
P921
P356
P1433
P1476
Macrophages and control of granulomatous inflammation in tuberculosis.
@en
P2093
P2860
P2888
P304
P356
10.1038/MI.2011.14
P577
2011-03-23T00:00:00Z