The switch from pRb/p105 to Rb2/p130 in DNA damage and cellular senescence. - Wikidata - wikimedia - TriplyDB

The switch from pRb/p105 to Rb2/p130 in DNA damage and cellular senescence.

The switch from pRb/p105 to Rb2/p130 in DNA damage and cellular senescence.

http://www.wikidata.org/entity/Q37861628

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Cell senescence in myxoid/round cell liposarcoma Hypoxia induces an undifferentiated phenotype of oral keratinocytes in vitro Senescence suppressors: their practical importance in replicative lifespan extension in stem cells.Tumour suppressors and cellular senescence.How does ethanol induce apoptotic cell death of SK-N-SH neuroblastoma cells.Inorganic arsenic exposure induces E2F-dependent G0/G1 arrest via an increase in retinoblastoma family protein p130 in B-cell lymphoma A20 cells.Dissecting Pin1 and phospho-pRb regulation.Misidentified Human Gene Functions with Mouse Models: The Case of the Retinoblastoma Gene Family in Senescence.Mesenchymal stromal cells having inactivated RB1 survive following low irradiation and accumulate damaged DNA: Hints for side effects following radiotherapy.Gene expression profiling identifies different sub-types of retinoblastoma.Adipose-Derived Mesenchymal Stem Cells from the Elderly Exhibit Decreased Migration and Differentiation Abilities with Senescent Properties.Timing of transcription during the cell cycle: Protein complexes binding to E2F, E2F/CLE, CDE/CHR, or CHR promoter elements define early and late cell cycle gene expression.Silencing of RB1 but not of RB2/P130 induces cellular senescence and impairs the differentiation potential of human mesenchymal stem cells.

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P2860

The switch from pRb/p105 to Rb2/p130 in DNA damage and cellular senescence.

http://www.wikidata.org/entity/Q37861628

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on February 2012

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

The switch from pRb/p105 to Rb2/p130 in DNA damage and cellular senescence.

@en

The switch from pRb/p105 to Rb2/p130 in DNA damage and cellular senescence.

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type

label

The switch from pRb/p105 to Rb2/p130 in DNA damage and cellular senescence.

@en

The switch from pRb/p105 to Rb2/p130 in DNA damage and cellular senescence.

@nl

prefLabel

The switch from pRb/p105 to Rb2/p130 in DNA damage and cellular senescence.

@en

The switch from pRb/p105 to Rb2/p130 in DNA damage and cellular senescence.

@nl

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Journal of Cellular Physiology

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The switch from pRb/p105 to Rb2/p130 in DNA damage and cellular senescence.

@en

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Heike Helmbold

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Umberto Galderisi

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Wolfgang Bohn

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P2860

MDC1 is a mediator of the mammalian DNA damage checkpoint

ATM and ATR substrate analysis reveals extensive protein networks responsive to DNA damage

Growth suppression by p16ink4 requires functional retinoblastoma protein

Negative regulation of DNA replication by the retinoblastoma protein is mediated by its association with MCM7.

E2F and histone deacetylase mediate transforming growth factor beta repression of cdc25A during keratinocyte cell cycle arrest

The three members of the pocket proteins family share the ability to repress E2F activity through recruitment of a histone deacetylase

E2F integrates cell cycle progression with DNA repair, replication, and G(2)/M checkpoints

Crucial role of p53-dependent cellular senescence in suppression of Pten-deficient tumorigenesis

Differential assembly of Cdc45p and DNA polymerases at early and late origins of DNA replication

CDK inhibitors: positive and negative regulators of G1-phase progression

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508-513

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scholarly article

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10.1002/JCP.22786

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2

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227

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2012-02-01T00:00:00Z

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21465484

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