Branched-chain amino acids as pharmacological nutrients in chronic liver disease. - Wikidata - wikimedia - TriplyDB

Branched-chain amino acids as pharmacological nutrients in chronic liver disease.

Branched-chain amino acids as pharmacological nutrients in chronic liver disease.

http://www.wikidata.org/entity/Q37874555

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Chemoprevention of obesity-related liver carcinogenesis by using pharmaceutical and nutraceutical agents Liver Cirrhosis: Evaluation, Nutritional Status, and Prognosis.Nutrition and metabolism in hepatocellular carcinoma Meta-analysis: oral or enteral nutritional supplementation in cirrhosis Supportive therapies for prevention of hepatocellular carcinoma recurrence and preservation of liver function Serum hyaluronic acid predicts protein-energy malnutrition in chronic hepatitis C.Serum albumin level is a notable profiling factor for non-B, non-C hepatitis virus-related hepatocellular carcinoma: A data-mining analysis.An association between dietary habits and traffic accidents in patients with chronic liver disease: A data-mining analysis.Branched-chain amino acids prevent hepatic fibrosis and development of hepatocellular carcinoma in a non-alcoholic steatohepatitis mouse model.A switch in the source of ATP production and a loss in capacity to perform glycolysis are hallmarks of hepatocyte failure in advance liver disease.Effect of psoas muscle mass after endoscopic therapy for patients with esophageal varices Protein tolerance to standard and high protein meals in patients with liver cirrhosis Effects of branched-chain amino acids (BCAAs) on the progression of advanced liver disease: A Korean nationwide, multicenter, retrospective, observational, cohort study.Comparison of Prognostic Impact between the Child-Pugh Score and Skeletal Muscle Mass for Patients with Liver Cirrhosis.Impact of Virtual Touch Quantification in Acoustic Radiation Force Impulse for Skeletal Muscle Mass Loss in Chronic Liver Diseases Management of hepatic encephalopathy in the hospital Branched-chain amino acid metabolism: from rare Mendelian diseases to more common disorders.Alterations of the gut microbiome and metabolome in alcoholic liver disease Prognostic significance of nonprotein respiratory quotient in patients with liver cirrhosis.Diet-induced hyperinsulinemia differentially affects glucose and protein metabolism: a high-throughput metabolomic approach in rats.Nutraceutical approach for preventing obesity-related colorectal and liver carcinogenesis.Branched-chain amino acid supplementation reduces oxidative stress and prolongs survival in rats with advanced liver cirrhosis Branched-chain amino acids ameliorate fibrosis and suppress tumor growth in a rat model of hepatocellular carcinoma with liver cirrhosis.Bolus ingestion of individual branched-chain amino acids alters plasma amino acid profiles in young healthy men Metformin suppresses diethylnitrosamine-induced liver tumorigenesis in obese and diabetic C57BL/KsJ-+Leprdb/+Leprdb mice.Branched-chain amino acids reduce hepatic iron accumulation and oxidative stress in hepatitis C virus polyprotein-expressing mice Plasma Amino Acid Concentrations Predict Mortality in Patients with End-Stage Liver Disease.Factors Associated With Protein-energy Malnutrition in Chronic Liver Disease: Analysis Using Indirect Calorimetry Nutritional status assessment in cirrhotic patients after protein supplementation.Effect of branched-chain amino acid-enriched nutritional supplementation on interferon therapy in Japanese patients with chronic hepatitis C virus infection: a retrospective study.Influence of insulin resistance on the development of hepatocellular carcinoma after antiviral treatment for non-cirrhotic patients with chronic hepatitis C.Effects of branched-chain amino acid granules on serum albumin level and prognosis are dependent on treatment adherence in patients with liver cirrhosis.The positive association of branched-chain amino acids and metabolic dyslipidemia in Chinese Han population.A targeted metabolomic protocol for short-chain fatty acids and branched-chain amino acids.Branched-chain amino acids suppress the cumulative recurrence of hepatocellular carcinoma under conditions of insulin-resistance Branched-chain amino acids in liver diseases.Cys34-cysteinylated human serum albumin is a sensitive plasma marker in oxidative stress-related chronic diseases.Effect of branched-chain amino acids in patients receiving intervention for hepatocellular carcinoma Infectious burden and atherosclerosis: A clinical issue.Hepatic arterial infusion chemotherapy for advanced hepatocellular carcinoma and future treatments for the poor responders.

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Branched-chain amino acids as pharmacological nutrients in chronic liver disease.

http://www.wikidata.org/entity/Q37874555

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on 23 June 2011

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

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name

Branched-chain amino acids as pharmacological nutrients in chronic liver disease.

@en

Branched-chain amino acids as pharmacological nutrients in chronic liver disease.

@nl

type

label

Branched-chain amino acids as pharmacological nutrients in chronic liver disease.

@en

Branched-chain amino acids as pharmacological nutrients in chronic liver disease.

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prefLabel

Branched-chain amino acids as pharmacological nutrients in chronic liver disease.

@en

Branched-chain amino acids as pharmacological nutrients in chronic liver disease.

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Branched-chain amino acids as pharmacological nutrients in chronic liver disease.

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Michael R Charlton

http://www.w3.org/2001/XMLSchema#string

Michio Sata

http://www.w3.org/2001/XMLSchema#string

Namiki Izumi

http://www.w3.org/2001/XMLSchema#string

Takumi Kawaguchi

http://www.w3.org/2001/XMLSchema#string

P2860

Hepatitis C Virus Down-Regulates Insulin Receptor Substrates 1 and 2 through Up-Regulation of Suppressor of Cytokine Signaling 3

Disruption of BCATm in mice leads to increased energy expenditure associated with the activation of a futile protein turnover cycle

Regional differences in cerebral blood flow and cerebral ammonia metabolism in patients with cirrhosis.

The IGF axis and hepatocarcinogenesis.

Factors associated with poor health-related quality of life of patients with cirrhosis.

Insulin-activated protein kinase Cbeta bypasses Ras and stimulates mitogen-activated protein kinase activity and cell proliferation in muscle cells.

Murine cirrhosis induces hepatocyte epithelial mesenchymal transition and alterations in survival signaling pathways.

Ammonia metabolism, the brain and fatigue; revisiting the link.

ESPEN Guidelines on Enteral Nutrition: Liver disease.

Insulin, insulin-like growth factor-I (IGF-I), IGF binding proteins, their biologic interactions, and colorectal cancer.

P304

1063-1070

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scholarly article

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10.1002/HEP.24412

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3

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54

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2011-06-23T00:00:00Z

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51118089

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21563202

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