A switch in the source of ATP production and a loss in capacity to perform glycolysis are hallmarks of hepatocyte failure in advance liver disease.
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Liver-Regenerative Transplantation: Regrow and ResetPrognostic significance of nonprotein respiratory quotient in patients with liver cirrhosis.High Concentrations of Atmospheric Ammonia Induce Alterations in the Hepatic Proteome of Broilers (Gallus gallus): An iTRAQ-Based Quantitative Proteomic AnalysisLack of the Lysosomal Membrane Protein, GLMP, in Mice Results in Metabolic Dysregulation in LiverChronic Intake of Japanese Sake Mediates Radiation-Induced Metabolic Alterations in Mouse LiverNitric oxide is a positive regulator of the Warburg effect in ovarian cancer cellsProteomics-Based Identification of the Molecular Signatures of Liver Tissues from Aged Rats following Eight Weeks of Medium-Intensity ExerciseMetabolomic mechanisms of gypenoside against liver fibrosis in rats: An integrative analysis of proteomics and metabolomics dataDysfunctional autophagy in RPE, a contributing factor in age-related macular degeneration.Regulation of hepatocyte identity and quiescence.Mechanisms of hepatotoxicity associated with the monocyclic β-lactam antibiotic BAL30072.Deregulation of energy metabolism promotes antifibrotic effects in human hepatic stellate cells and prevents liver fibrosis in a mouse model.Mitochondrial bioenergetics and posthepatectomy liver dysfunction.Down-regulation of oxidative phosphorylation in the liver by expression of the ATPase inhibitory factor 1 induces a tumor-promoter metabolic stateThe association of early combined lactate and glucose levels with subsequent renal and liver dysfunction and hospital mortality in critically ill patients.Metabolomics and eicosanoid analysis identified serum biomarkers for distinguishing hepatocellular carcinoma from hepatitis B virus-related cirrhosis.Ordinary differential equations and Boolean networks in application to modelling of 6-mercaptopurine metabolism.Reply to: "Is the pathway of energy metabolism modified in advanced cirrhosis?".Melatonin protects against lipid-induced mitochondrial dysfunction in hepatocytes and inhibits stellate cell activation during hepatic fibrosis in mice.Elevated mitochondrial activity distinguishes fibrogenic hepatic stellate cells and sensitizes for selective inhibition by mitotropic doxorubicin.Salvianolate reduces glucose metabolism disorders in dimethylnitrosamine-induced cirrhotic rats.Profiling of the circadian metabolome in thioacetamide-induced liver cirrhosis in mice.A Case of Levofloxacin-Induced Hepatotoxicity.Liver-enriched transcription factor expression relates to chronic hepatic failure in humans.Immunometabolism: A novel perspective of liver cancer microenvironment and its influence on tumor progressionHigh Expression of Glycolytic Genes in Cirrhosis Correlates With the Risk of Developing Liver Cancer
P2860
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P2860
A switch in the source of ATP production and a loss in capacity to perform glycolysis are hallmarks of hepatocyte failure in advance liver disease.
description
2014 nî lūn-bûn
@nan
2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
A switch in the source of ATP ...... lure in advance liver disease.
@ast
A switch in the source of ATP ...... lure in advance liver disease.
@en
type
label
A switch in the source of ATP ...... lure in advance liver disease.
@ast
A switch in the source of ATP ...... lure in advance liver disease.
@en
prefLabel
A switch in the source of ATP ...... lure in advance liver disease.
@ast
A switch in the source of ATP ...... lure in advance liver disease.
@en
P2093
P2860
P50
P1476
A switch in the source of ATP ...... lure in advance liver disease.
@en
P2093
Aaron Damm
Alejandro Soto-Gutierrez
Iraklis I Pipinos
Michael Oertel
Mladen I Yovchev
Taichiro Nishikawa
Vasudha Sehgal
P2860
P304
P356
10.1016/J.JHEP.2014.02.014
P577
2014-02-26T00:00:00Z