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Programmed necrosis in acute kidney injuryTWEAK and the progression of renal disease: clinical translationLupus nephritis: the evolving role of novel therapeuticsNF-κB in inflammation and renal diseasesTWEAK promotes peritoneal inflammationSoluble Fn14 Is Detected and Elevated in Mouse and Human Kidney DiseaseInflammatory cytokines and survival factors from serum modulate tweak-induced apoptosis in PC-3 prostate cancer cells.TWEAK/Fn14 system and crescent formation in IgA nephropathy.Deficiency of fibroblast growth factor-inducible 14 (Fn14) preserves the filtration barrier and ameliorates lupus nephritis.Putative novel mediators of acute kidney injury in critically ill patients: handling by continuous venovenous hemofiltration and effect of anticoagulation modalitiesOne dose of cyclosporine A is protective at initiation of folic acid-induced acute kidney injury in miceInhibition of the TWEAK/Fn14 pathway attenuates renal disease in nephrotoxic serum nephritisTumor Necrosis Factor-Like Weak Inducer of Apoptosis Accelerates the Progression of Renal Fibrosis in Lupus Nephritis by Activating SMAD and p38 MAPK in TGF-β1 Signaling PathwayTwo independent pathways of regulated necrosis mediate ischemia-reperfusion injuryThe RIP1-kinase inhibitor necrostatin-1 prevents osmotic nephrosis and contrast-induced AKI in mice.TWEAK/Fn14 pathway is a novel mediator of retinal neovascularization.Urine biomarkers in juvenile-onset SLE nephritis.The TWEAK/Fn14 pathway as an aggravating and perpetuating factor in inflammatory diseases: focus on inflammatory bowel diseases.Cardiovascular risk biomarkers in CKD: the inflammation link and the road less traveled.A further TWEAK to multiple sclerosis pathophysiology.TWEAK/Fn14 and Non-Canonical NF-kappaB Signaling in Kidney DiseaseTWEAK: A New Player in Obesity and Diabetes.Mitochondria-targeted therapies for acute kidney injury.Sustained remission in lupus nephritis: still a hard road ahead.Role of the TWEAK/Fn14 pathway in autoimmune diseases.TNF-related weak inducer of apoptosis (TWEAK) regulates junctional proteins in tubular epithelial cells via canonical NF-κB pathway and ERK activation.Mitogen-Activated Protein Kinase 14 Promotes AKI.Histone lysine crotonylation during acute kidney injury in miceNF-κB inducing kinase is a therapeutic target for systemic lupus erythematosus.AKI on CKD: heightened injury, suppressed repair, and the underlying mechanisms.p-cresyl sulphate has pro-inflammatory and cytotoxic actions on human proximal tubular epithelial cells.TWEAK-Fn14 Influences Neurogenesis Status via Modulating NF-κB in Mice with Spinal Cord Injury.Tnfrsf12a-Mediated Atherosclerosis Signaling and Inflammatory Response as a Common Protection Mechanism of Shuxuening Injection Against Both Myocardial and Cerebral Ischemia-Reperfusion Injuries.TWEAK increases CD74 expression and sensitizes to DDT proinflammatory actions in tubular cells.
P2860
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P2860
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 22 June 2011
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
TWEAK, a multifunctional cytokine in kidney injury.
@en
TWEAK, a multifunctional cytokine in kidney injury.
@nl
type
label
TWEAK, a multifunctional cytokine in kidney injury.
@en
TWEAK, a multifunctional cytokine in kidney injury.
@nl
prefLabel
TWEAK, a multifunctional cytokine in kidney injury.
@en
TWEAK, a multifunctional cytokine in kidney injury.
@nl
P50
P356
P1433
P1476
TWEAK, a multifunctional cytokine in kidney injury.
@en
P2888
P304
P356
10.1038/KI.2011.180
P407
P577
2011-06-22T00:00:00Z