In vivo conversion of adult α-cells into β-like cells: a new research avenue in the context of type 1 diabetes. - Wikidata - wikimedia - TriplyDB

In vivo conversion of adult α-cells into β-like cells: a new research avenue in the context of type 1 diabetes.

In vivo conversion of adult α-cells into β-like cells: a new research avenue in the context of type 1 diabetes.

http://www.wikidata.org/entity/Q37913506

about

β-cell replacement sources for type 1 diabetes: a focus on pancreatic ductal cells Crosstalk between the developing pancreas and its blood vessels: an evolving dialog.α-cell role in β-cell generation and regeneration.Evolving function and potential of pancreatic alpha cells.Non-invasive in vivo determination of viable islet graft volume by 111In-exendin-3.The temporal and hierarchical control of transcription factors-induced liver to pancreas transdifferentiation.Conversion of mature human β-cells into glucagon-producing α-cells.Pax4 is not essential for beta-cell differentiation in zebrafish embryos but modulates alpha-cell generation by repressing arx gene expression.Development and regeneration in the endocrine pancreas Ultrastructural alterations of pancreatic beta cells in human diabetes mellitus.

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In vivo conversion of adult α-cells into β-like cells: a new research avenue in the context of type 1 diabetes.

http://www.wikidata.org/entity/Q37913506

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In vivo conversion of adult α- ...... he context of type 1 diabetes.

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In vivo conversion of adult α- ...... he context of type 1 diabetes.

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In vivo conversion of adult α- ...... he context of type 1 diabetes.

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In vivo conversion of adult α- ...... he context of type 1 diabetes.

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J.1463-1326.2011.01441.X

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Diabetes, Obesity and Metabolism

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In vivo conversion of adult α- ...... he context of type 1 diabetes.

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A Pfeifer

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A Vieira

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E Gjernes

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M Courtney

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P2860

Involvement of the homeodomain-containing transcription factor PDX-1 in islet amyloid polypeptide gene transcription

The MafA transcription factor appears to be responsible for tissue-specific expression of insulin

The ectopic expression of Pax4 in the mouse pancreas converts progenitor cells into alpha and subsequently beta cells

neurogenin3 is required for the development of the four endocrine cell lineages of the pancreas

Positioning of five genes (CASK, ARX, SAT, IMAGE cDNAs 248928 and 253949) from the human X chromosome short arm with respect to evolutionary breakpoints on the mouse X chromosome

Autoregulation and maturity onset diabetes of the young transcription factors control the human PAX4 promoter

Independent requirement for ISL1 in formation of pancreatic mesenchyme and islet cells

Notch signalling controls pancreatic cell differentiation

The simultaneous loss of Arx and Pax4 genes promotes a somatostatin-producing cell fate specification at the expense of the alpha- and beta-cell lineages in the mouse endocrine pancreas

Lower blood glucose, hyperglucagonemia, and pancreatic alpha cell hyperplasia in glucagon receptor knockout mice

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47-52

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10.1111/J.1463-1326.2011.01441.X

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13 Suppl 1

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Patrick Collombat

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2011-10-01T00:00:00Z

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51554664

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21824256

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type-1 diabetes