Molecular insight into invasive group A streptococcal disease.
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Molecular epidemiology and genomics of group A StreptococcusVirulence of Group A Streptococci Is Enhanced by Human Complement InhibitorsIdentification of novel growth phase- and media-dependent small non-coding RNAs in Streptococcus pyogenes M49 using intergenic tiling arraysStructure-Informed Design of an Enzymatically Inactive Vaccine Component for Group A StreptococcusMolecular and genomic characterization of pathogenic traits of group A Streptococcus pyogenesRole for streptococcal collagen-like protein 1 in M1T1 group A Streptococcus resistance to neutrophil extracellular trapsEvolutionary Constraints Shaping Streptococcus pyogenes-Host Interactions.Tracing the evolutionary history of the pandemic group A streptococcal M1T1 clone.Complete Genome Sequence of emm1 Streptococcus pyogenes A20, a Strain with an Intact Two-Component System, CovRS, Isolated from a Patient with Necrotizing Fasciitis.Coiled-coil irregularities of the M1 protein structure promote M1-fibrinogen interaction and influence group A Streptococcus host cell interactions and virulence.Vaccination against the M protein of Streptococcus pyogenes prevents death after influenza virus: S. pyogenes super-infection.Infection-induced NETosis is a dynamic process involving neutrophil multitasking in vivo.The Abi-domain protein Abx1 interacts with the CovS histidine kinase to control virulence gene expression in group B Streptococcus.The innate immune response elicited by Group A Streptococcus is highly variable among clinical isolates and correlates with the emm type.Opacification Domain of Serum Opacity Factor Inhibits Beta-Hemolysis and Contributes to Virulence of Streptococcus pyogenes.Rapid Emergence of a New Clone Impacts the Population at Risk and Increases the Incidence of Type emm89 Group A Streptococcus Invasive Disease.Differences in SpeB protease activity among group A streptococci associated with superficial, invasive, and autoimmune disease.Streptococcal collagen-like protein A and general stress protein 24 are immunomodulating virulence factors of group A Streptococcus.A highly active and negatively charged Streptococcus pyogenes lysin with a rare D-alanyl-L-alanine endopeptidase activity protects mice against streptococcal bacteremia.Importance of whole genome sequencing for the assessment of outbreaks in diagnostic laboratories: analysis of a case series of invasive Streptococcus pyogenes infections.Endothelial cells are intrinsically defective in xenophagy of Streptococcus pyogenes.Molecular characterization of the 2011 Hong Kong scarlet fever outbreak.Essential Genes in the Core Genome of the Human Pathogen Streptococcus pyogenes.Inhibitor of streptokinase gene expression improves survival after group A streptococcus infection in miceHyaluronan breakdown contributes to immune defense against group A StreptococcusResponses of innate immune cells to group A Streptococcus.DNase Sda1 allows invasive M1T1 Group A Streptococcus to prevent TLR9-dependent recognitionStaphylococcal and streptococcal superantigen exotoxinsThe contribution of group A streptococcal virulence determinants to the pathogenesis of sepsis.Disease manifestations and pathogenic mechanisms of Group A Streptococcus.A bacterial pathogen co-opts host plasmin to resist killing by cathelicidin antimicrobial peptidesThe crimson conundrum: heme toxicity and tolerance in GAS.Horizontal gene transfer and recombination in Streptococcus dysgalactiae subsp. equisimilisThe FasX Small Regulatory RNA Negatively Regulates the Expression of Two Fibronectin-Binding Proteins in Group A Streptococcus.Mutual exclusivity of hyaluronan and hyaluronidase in invasive group A Streptococcus.Determining antibody-binding site of streptococcal pyrogenic exotoxin B to protect mice from group a streptococcus infectionUnique genomic arrangements in an invasive serotype M23 strain of Streptococcus pyogenes identify genes that induce hypervirulence.The AgI/II family adhesin AspA is required for respiratory infection by Streptococcus pyogenes.The human antimicrobial peptide LL-37 binds directly to CsrS, a sensor histidine kinase of group A Streptococcus, to activate expression of virulence factors.A novel role for pro-coagulant microvesicles in the early host defense against streptococcus pyogenes.
P2860
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P2860
Molecular insight into invasive group A streptococcal disease.
description
article científic
@ca
article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
@pt
artigo científico
@pt-br
artikel ilmiah
@id
artikull shkencor
@sq
artículo científico
@es
name
Molecular insight into invasive group A streptococcal disease.
@en
Molecular insight into invasive group A streptococcal disease.
@nl
type
label
Molecular insight into invasive group A streptococcal disease.
@en
Molecular insight into invasive group A streptococcal disease.
@nl
prefLabel
Molecular insight into invasive group A streptococcal disease.
@en
Molecular insight into invasive group A streptococcal disease.
@nl
P356
P1476
Molecular insight into invasive group A streptococcal disease.
@en
P2093
Jason N Cole
Timothy C Barnett
P2888
P304
P356
10.1038/NRMICRO2648
P407
P577
2011-09-16T00:00:00Z