Through a glass less darkly: apoptosis and the germinal center response to antigen. - Wikidata - wikimedia - TriplyDB

Through a glass less darkly: apoptosis and the germinal center response to antigen.

Through a glass less darkly: apoptosis and the germinal center response to antigen.

http://www.wikidata.org/entity/Q38002487

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Defects in Germinal Center Selection in SLE The AP-1 transcription factor Fra1 inhibits follicular B cell differentiation into plasma cells.Follicular dendritic cells in health and disease.EAF2 mediates germinal centre B-cell apoptosis to suppress excessive immune responses and prevent autoimmunity.MCL-1 is required throughout B-cell development and its loss sensitizes specific B-cell subsets to inhibition of BCL-2 or BCL-XL.Waldenström macroglobulinemia: clinical and immunological aspects, natural history, cell of origin, and emerging mouse models Transcription factors regulating B cell fate in the germinal centre.Murine models of germinal center derived-lymphomas.Functional disparities among BCL-2 members in tonsillar and leukemic B-cell subsets assessed by BH3-mimetic profiling.The life and death of immune cell types: the role of BCL-2 anti-apoptotic molecules.Intracellular BH3 Profiling Reveals Shifts in Antiapoptotic Dependency in Human B Cell Maturation and Mitogen-Stimulated Proliferation.Hyperactivated PI3Kδ promotes self and commensal reactivity at the expense of optimal humoral immunity Germinal Center B Cells Replace Their Antigen Receptors in Dark Zones and Fail Light Zone Entry when Immunoglobulin Gene Mutations are Damaging

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Through a glass less darkly: apoptosis and the germinal center response to antigen.

http://www.wikidata.org/entity/Q38002487

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Through a glass less darkly: apoptosis and the germinal center response to antigen.

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Through a glass less darkly: apoptosis and the germinal center response to antigen.

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Through a glass less darkly: apoptosis and the germinal center response to antigen.

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J.1600-065X.2012.01123.X

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Immunological Reviews

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Through a glass less darkly: apoptosis and the germinal center response to antigen.

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David M Tarlinton

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Ingela B Vikstrom

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Victor Peperzak

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MCL-1S, a splicing variant of the antiapoptotic BCL-2 family member MCL-1, encodes a proapoptotic protein possessing only the BH3 domain

Mule/ARF-BP1, a BH3-only E3 ubiquitin ligase, catalyzes the polyubiquitination of Mcl-1 and regulates apoptosis

Glycogen synthase kinase-3 regulates mitochondrial outer membrane permeabilization and apoptosis by destabilization of MCL-1

Mechanisms of apoptosis sensitivity and resistance to the BH3 mimetic ABT-737 in acute myeloid leukemia

MCL-1ES, a novel variant of MCL-1, associates with MCL-1L and induces mitochondrial cell death

Selective generation of functional somatically mutated IgM+CD27+, but not Ig isotype-switched, memory B cells in X-linked lymphoproliferative disease

Transcriptional analysis of the B cell germinal center reaction

MCL1, a gene expressed in programmed myeloid cell differentiation, has sequence similarity to BCL2

Molecular subtypes of diffuse large B-cell lymphoma arise by distinct genetic pathways

Transcription enhances AID-mediated cytidine deamination by exposing single-stranded DNA on the nontemplate strand

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93-106

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10.1111/J.1600-065X.2012.01123.X

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1

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247

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2012-05-01T00:00:00Z

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22500834

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