The complex regulation of TGF-β in cardiovascular disease. - Wikidata - wikimedia - TriplyDB

The complex regulation of TGF-β in cardiovascular disease.

The complex regulation of TGF-β in cardiovascular disease.

http://www.wikidata.org/entity/Q38048775

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Delayed administration of allogeneic cardiac stem cell therapy for acute myocardial infarction could ameliorate adverse remodeling: experimental study in swine.Association of Plasma Transforming Growth Factor-β1 Levels and the Risk of Atrial Fibrillation: A Meta-Analysis Low-dose memantine attenuated methadone dose in opioid-dependent patients: a 12-week double-blind randomized controlled trial.Role of caveolin-1 in atrial fibrillation as an anti-fibrotic signaling molecule in human atrial fibroblasts.Age-dependent defective TGF-beta1 signaling in patients undergoing coronary artery bypass grafting.RGS3 inhibits TGF-β1/Smad signalling in adventitial fibroblasts.A state of reversible compensated ventricular dysfunction precedes pathological remodelling in response to cardiomyocyte-specific activity of angiotensin II type-1 receptor in mice.The Relationship between VEGFA and TGFB1 Polymorphisms and Target Lesion Revascularization after Elective Percutaneous Coronary Intervention Elevated circulating TGF-β is not the cause of increased atherosclerosis development in biglycan deficient mice.

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The complex regulation of TGF-β in cardiovascular disease.

http://www.wikidata.org/entity/Q38048775

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The complex regulation of TGF-β in cardiovascular disease.

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The complex regulation of TGF-β in cardiovascular disease.

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The complex regulation of TGF-β in cardiovascular disease.

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Vascular Health and Risk Management

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The complex regulation of TGF-β in cardiovascular disease.

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Jorge Navarro-Dorado

http://www.w3.org/2001/XMLSchema#string

Marta Ramajo

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Santiago Redondo

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Teresa Tejerina

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Úrsula Medina

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P2860

Cholesterol suppresses cellular TGF-beta responsiveness: implications in atherogenesis

Activin receptor-like kinase (ALK)1 is an antagonistic mediator of lateral TGFbeta/ALK5 signaling

Nuclear targeting of transforming growth factor-beta-activated Smad complexes

Endoglin, a TGF-beta binding protein of endothelial cells, is the gene for hereditary haemorrhagic telangiectasia type 1

Increased plasma soluble endoglin levels as an indicator of cardiovascular alterations in hypertensive and diabetic patients

Targeted deletion reveals essential and overlapping functions of the miR-17 through 92 family of miRNA clusters

The activin receptor-like kinase 1 gene: genomic structure and mutations in hereditary hemorrhagic telangiectasia type 2

The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1

Caveolin-1 regulates transforming growth factor (TGF)-beta/SMAD signaling through an interaction with the TGF-beta type I receptor

Acetylsalicylic acid inhibits cell proliferation by involving transforming growth factor-beta

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533-539

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scholarly article

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10.2147/VHRM.S28041

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8

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2012-09-13T00:00:00Z

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231742846

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23028232

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Aparato circulatorio

cardiovascular disease

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3446857

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