Targeting c-FLICE-like inhibitory protein (CFLAR) in cancer. - Wikidata - wikimedia - TriplyDB

Targeting c-FLICE-like inhibitory protein (CFLAR) in cancer.

Targeting c-FLICE-like inhibitory protein (CFLAR) in cancer.

http://www.wikidata.org/entity/Q38068479

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Marine Drugs Regulating Apoptosis Induced by Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand (TRAIL)FADD regulates NF-κB activation and promotes ubiquitination of cFLIPL to induce apoptosis Generalized monotone incremental forward stagewise method for modeling count data: application predicting micronuclei frequency.Roles of c-FLIP in Apoptosis, Necroptosis, and Autophagy.c-FLIP is involved in tumor progression of peripheral T-cell lymphoma and targeted by histone deacetylase inhibitors.Multiple mechanisms mediate resistance to sorafenib in urothelial cancer Dysregulated signaling, proliferation and apoptosis impact on the pathogenesis of TCRγδ+ T cell large granular lymphocyte leukemia.Inhibition of c-FLIPL expression by miRNA-708 increases the sensitivity of renal cancer cells to anti-cancer drugs.Icaritin Sensitizes Human Glioblastoma Cells to TRAIL-Induced Apoptosis.Correlation of glucocorticoid-mediated E4BP4 upregulation with altered expression of pro- and anti-apoptotic genes in CEM human lymphoblastic leukemia cells.Luteolin induces apoptosis by activating Fas signaling pathway at the receptor level in laryngeal squamous cell line Hep-2 cells.Regulation of cell death in cancer-possible implications for immunotherapy.Glycogen synthase kinase-3β inhibition promotes lysosome-dependent degradation of c-FLIPL in hepatocellular carcinoma.HDAC and Ku70 axis- an effective target for apoptosis induction by a new 2-cyano-3-oxo-1,9-dien glycyrrhetinic acid analogue.

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Targeting c-FLICE-like inhibitory protein (CFLAR) in cancer.

http://www.wikidata.org/entity/Q38068479

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article científic

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artikel ilmiah

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Targeting c-FLICE-like inhibitory protein (CFLAR) in cancer.

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Targeting c-FLICE-like inhibitory protein (CFLAR) in cancer.

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Targeting c-FLICE-like inhibitory protein (CFLAR) in cancer.

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14728222.2013.736499

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Expert Opinion on Therapeutic Targets

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Targeting c-FLICE-like inhibitory protein (CFLAR) in cancer.

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Bortezomib primes glioblastoma, including glioblastoma stem cells, for TRAIL by increasing tBid stability and mitochondrial apoptosis

cIAPs block Ripoptosome formation, a RIP1/caspase-8 containing intracellular cell death complex differentially regulated by cFLIP isoforms

Activation of the NF-kappaB pathway by caspase 8 and its homologs

The caspase-8 inhibitor FLIP promotes activation of NF-kappaB and Erk signaling pathways

The Akt-regulated forkhead transcription factor FOXO3a controls endothelial cell viability through modulation of the caspase-8 inhibitor FLIP

The long form of FLIP is an activator of caspase-8 at the Fas death-inducing signaling complex

Cotreatment with histone deacetylase inhibitor LAQ824 enhances Apo-2L/tumor necrosis factor-related apoptosis inducing ligand-induced death inducing signaling complex activity and apoptosis of human acute leukemia cells

The c-MYC oncoprotein is a substrate of the acetyltransferases hGCN5/PCAF and TIP60

Catalytic activity of the caspase-8-FLIP(L) complex inhibits RIPK3-dependent necrosis

The E3 ubiquitin ligase itch couples JNK activation to TNFalpha-induced cell death by inducing c-FLIP(L) turnover

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195-201

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scholarly article

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10.1517/14728222.2013.736499

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