Postsynaptic Receptors for Amyloid-β Oligomers as Mediators of Neuronal Damage in Alzheimer's Disease.
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The Role of Amyloid-β Oligomers in Toxicity, Propagation, and ImmunotherapyThe dendritic hypothesis for Alzheimer's disease pathophysiology.Alzheimer's disease: relevant molecular and physiopathological events affecting amyloid-β brain balance and the putative role of PPARs.The intersection of amyloid beta and tau at synapses in Alzheimer's diseaseSmall-molecule modulation of neurotrophin receptors: a strategy for the treatment of neurological disease.Dextran sulfate sodium inhibits amyloid-β oligomer binding to cellular prion protein.Emerging Synaptic Molecules as Candidates in the Etiology of Neurological Disorders.Overcoming barriers and thresholds - signaling of oligomeric Aβ through the prion protein to Fyn.Progress towards a consensus on biomarkers for Alzheimer's disease: a review of peripheral analytes.Role of Sirt1 during the ageing process: relevance to protection of synapses in the brain.Posttranslational Modifications Regulate the Postsynaptic Localization of PSD-95.Non-Demented Individuals with Alzheimer's Disease Neuropathology: Resistance to Cognitive Decline May Reveal New Treatment Strategies.Wnt-related SynGAP1 is a neuroprotective factor of glutamatergic synapses against Aβ oligomers.Preclinical to phase II amyloid beta (Aβ) peptide modulators under investigation for Alzheimer's disease.PPARs in the central nervous system: roles in neurodegeneration and neuroinflammation.Wnt/TLR Dialog in Neuroinflammation, Relevance in Alzheimer's Disease.The transcription factor XBP1 in memory and cognition: Implications in Alzheimer disease.The neuronal membrane as a key factor in neurodegeneration.Intracellular oligomeric amyloid-beta rapidly regulates GluA1 subunit of AMPA receptor in the hippocampus.Inhibition of group-I metabotropic glutamate receptors protects against prion toxicity.NSCs promote hippocampal neurogenesis, metabolic changes and synaptogenesis in APP/PS1 transgenic mice.Nicotine prevents synaptic impairment induced by amyloid-β oligomers through α7-nicotinic acetylcholine receptor activation.Therapeutics of Neurotransmitters in Alzheimer's Disease.Unresolved questions in Alzheimer's research: will biomarkers help?NMDA Receptor Subunits Change after Synaptic Plasticity Induction and Learning and Memory Acquisition.Phosphorylation of the Amyloid-Beta Peptide Inhibits Zinc-Dependent Aggregation, Prevents Na,K-ATPase Inhibition, and Reduces Cerebral Plaque Deposition
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P2860
Postsynaptic Receptors for Amyloid-β Oligomers as Mediators of Neuronal Damage in Alzheimer's Disease.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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artigo científico
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artigo científico
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Postsynaptic Receptors for Amy ...... Damage in Alzheimer's Disease.
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type
label
Postsynaptic Receptors for Amy ...... Damage in Alzheimer's Disease.
@en
prefLabel
Postsynaptic Receptors for Amy ...... Damage in Alzheimer's Disease.
@en
P2093
P2860
P356
P1476
Postsynaptic Receptors for Amy ...... Damage in Alzheimer's Disease.
@en
P2093
Juvenal A Ríos
Margarita C Dinamarca
Nibaldo C Inestrosa
P2860
P356
10.3389/FPHYS.2012.00464
P577
2012-12-20T00:00:00Z