Role of the Rho GTPase Rac in the activation of the phagocyte NADPH oxidase: outsourcing a key task.
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NADPH oxidases as electrochemical generators to produce ion fluxes and turgor in fungi, plants and humansNADPH oxidases: key modulators in aging and age-related cardiovascular diseases?Reactive oxygen species: players in the cardiovascular effects of testosteroneOptical Tools To Study the Isoform-Specific Roles of Small GTPases in Immune Cells.Oxidative stress responses in the human fungal pathogen, Candida albicans.Nox Inhibitors & Therapies: Rational Design of Peptidic and Small Molecule Inhibitors5d, a novel analogue of 3-n-butylphthalide, decreases NADPH oxidase activity through the positive regulation of CK2 after ischemia/reperfusion injury.NADPH oxidase in brain injury and neurodegenerative disordersNox and renal disease.Regulation of NADPH oxidases in skeletal muscle.Dynasore enhances the formation of mitochondrial antiviral signalling aggregates and endocytosis-independent NF-κB activationExposure to chronic hyperglycemic conditions results in Ras-related C3 botulinum toxin substrate 1 (Rac1)-mediated activation of p53 and ATM kinase in pancreatic β-cells.Targeting reactive oxygen species in development and progression of pancreatic cancer.Rho GTPases at the crossroad of signaling networks in mammals.Knock-down of farnesyl pyrophosphate synthase protects heart-derived H9c2 cells against hypoxia/reoxygenation-induced injury.Efficacy of N-Acetylserotonin and Melatonin in the EAE Model of Multiple Sclerosis.Vascular injury in diabetic db/db mice is ameliorated by atorvastatin: role of Rac1/2-sensitive Nox-dependent pathways.ROP GTPases Structure-Function and Signaling Pathways.NADPH Oxidase Deficiency: A Multisystem Approach.shRNA-induced silencing of Ras-related C3 botulinum toxin substrate 1 inhibits the proliferation of colon cancer cells through upregulation of BAD and downregulation of cyclin D1.Pathogenic Effects of IFIT2 and Interferon-β during Fatal Systemic Candida albicans Infection.N-Formyl Peptide Receptors Induce Radical Oxygen Production in Fibroblasts Derived From Systemic Sclerosis by Interacting With a Cleaved Form of Urokinase Receptor.[NADPH oxidase: double agent during inflammation?]
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Role of the Rho GTPase Rac in the activation of the phagocyte NADPH oxidase: outsourcing a key task.
description
article científic
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article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
@pt
artigo científico
@pt-br
artikel ilmiah
@id
artikull shkencor
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artículo científico
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name
Role of the Rho GTPase Rac in ...... idase: outsourcing a key task.
@en
type
label
Role of the Rho GTPase Rac in ...... idase: outsourcing a key task.
@en
prefLabel
Role of the Rho GTPase Rac in ...... idase: outsourcing a key task.
@en
P2860
P356
P1476
Role of the Rho GTPase Rac in ...... idase: outsourcing a key task.
@en
P2093
Edgar Pick
P2860
P304
P356
10.4161/SGTP.27952
P50
P577
2014-03-05T00:00:00Z