The myosin-activated thin filament regulatory state, M⁻-open: a link to hypertrophic cardiomyopathy (HCM).
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Cardiac MyBP-C regulates the rate and force of contraction in mammalian myocardiumContributions of Ca2+-Independent Thin Filament Activation to Cardiac Muscle Function.N-acetylcysteine reverses diastolic dysfunction and hypertrophy in familial hypertrophic cardiomyopathy.Structural determinants of muscle thin filament cooperativity.The cMyBP-C HCM variant L348P enhances thin filament activation through an increased shift in tropomyosin position.Order-Disorder Transitions in the Cardiac Troponin Complex.The Relaxation Properties of Myofibrils Are Compromised by Amino Acids that Stabilize α-Tropomyosin.Cross-talk, cross-bridges, and calcium activation of cardiac contraction.
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The myosin-activated thin filament regulatory state, M⁻-open: a link to hypertrophic cardiomyopathy (HCM).
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article científic
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article scientifique
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articol științific
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articolo scientifico
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artigo científico
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artigo científico
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artigo científico
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artikel ilmiah
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artikull shkencor
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artículo científico
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name
The myosin-activated thin fila ...... rtrophic cardiomyopathy (HCM).
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type
label
The myosin-activated thin fila ...... rtrophic cardiomyopathy (HCM).
@en
prefLabel
The myosin-activated thin fila ...... rtrophic cardiomyopathy (HCM).
@en
P2860
P1476
The myosin-activated thin fila ...... ertrophic cardiomyopathy (HCM)
@en
P2093
Sherwin S Lehrer
P2860
P2888
P304
P356
10.1007/S10974-014-9383-Z
P577
2014-04-17T00:00:00Z
P5875
P6179
1039746425