IL-1β dependent cerebellar synaptopathy in a mouse mode of multiple sclerosis.
about
Altered excitatory-inhibitory balance within somatosensory cortex is associated with enhanced plasticity and pain sensitivity in a mouse model of multiple sclerosis.Glutamate, T cells and multiple sclerosis.Astrocyte matricellular proteins that control excitatory synaptogenesis are regulated by inflammatory cytokines and correlate with paralysis severity during experimental autoimmune encephalomyelitis.Inflammatory demyelination alters subcortical visual circuits.A novel crosstalk within the endocannabinoid system controls GABA transmission in the striatum.Changes of synapses in experimental autoimmune encephalomyelitis by using Fasudil.Tumor Necrosis Factor and Interleukin-1β Modulate Synaptic Plasticity during Neuroinflammation.
P2860
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P2860
IL-1β dependent cerebellar synaptopathy in a mouse mode of multiple sclerosis.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年学术文章
@wuu
2015年学术文章
@zh-cn
2015年学术文章
@zh-hans
2015年学术文章
@zh-my
2015年学术文章
@zh-sg
2015年學術文章
@yue
2015年學術文章
@zh
2015年學術文章
@zh-hant
name
IL-1β dependent cerebellar synaptopathy in a mouse mode of multiple sclerosis.
@en
type
label
IL-1β dependent cerebellar synaptopathy in a mouse mode of multiple sclerosis.
@en
prefLabel
IL-1β dependent cerebellar synaptopathy in a mouse mode of multiple sclerosis.
@en
P2860
P50
P1433
P1476
IL-1β dependent cerebellar synaptopathy in a mouse mode of multiple sclerosis.
@en
P2860
P2888
P356
10.1007/S12311-014-0613-0
P577
2015-02-01T00:00:00Z
P5875
P6179
1033297707