Hypoxia enhances the expression of autocrine motility factor and the motility of human pancreatic cancer cells - Wikidata - wikimedia - TriplyDB

Hypoxia enhances the expression of autocrine motility factor and the motility of human pancreatic cancer cells

Hypoxia enhances the expression of autocrine motility factor and the motility of human pancreatic cancer cells

http://www.wikidata.org/entity/Q38283699

about

Hypoxia-inducible factor 1 and cardiovascular disease Differential expression of growth factors at the cellular level in virus-infected brain The role of hypoxia inducible factor-1 in hepatocellular carcinoma.Microenvironmental and cellular consequences of altered blood flow in tumours.Autophagy in pancreatic cancer pathogenesis and treatment Microfluidics‑based optimization of neuroleukin‑mediated regulation of articular chondrocyte proliferation Development of novel therapeutic strategies that target HIF-1.sMEK1 inhibits endothelial cell proliferation by attenuating VEGFR-2-dependent-Akt/eNOS/HIF-1α signaling pathways.Hypoxia-induced endoplasmic reticulum stress characterizes a necrotic phenotype of pancreatic cancer.Hypoxia and angiogenesis in pancreatic cancer.Hypoxia-mediated up-regulation of Pim-1 contributes to solid tumor formation.Metabolic transformation in cancer.Glucose metabolism gene polymorphisms and clinical outcome in pancreatic cancer.Glucose metabolism gene variants modulate the risk of pancreatic cancer.Lipid rafts: signaling and sorting platforms of cells and their roles in cancer.Lactate as an insidious metabolite due to the Warburg effect.Targeting cancer cell metabolism in pancreatic adenocarcinoma.Metabolic changes associated with tumor metastasis, part 1: tumor pH, glycolysis and the pentose phosphate pathway.Glucose metabolic phenotype of pancreatic cancer.The neurotrophic factor neurturin contributes toward an aggressive cancer cell phenotype, neuropathic pain and neuronal plasticity in pancreatic cancer.Targeting the Metabolic Reprogramming That Controls Epithelial-to-Mesenchymal Transition in Aggressive Tumors.Pim-1 plays a pivotal role in hypoxia-induced chemoresistance.Cellular differentiation determines the expression of the hypoxia-inducible protein NDRG1 in pancreatic cancer.Differential regulation of phosphoglucose isomerase/autocrine motility factor activities by protein kinase CK2 phosphorylation.Metabolic Plasiticy in Cancers-Distinct Role of Glycolytic Enzymes GPI, LDHs or Membrane Transporters MCTs.Role of TRPC1 channels in pressure-mediated activation of murine pancreatic stellate cells.Higher autocrine motility factor/glucose-6-phosphate isomerase expression is associated with tumorigenesis and poorer prognosis in gastric cancer

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P2860

Hypoxia enhances the expression of autocrine motility factor and the motility of human pancreatic cancer cells

http://www.wikidata.org/entity/Q38283699

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Hypoxia enhances the expressio ...... human pancreatic cancer cells

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A Raz

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H Katoh

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H Niizeki

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H Watanabe

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M Hosokawa

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M Kobayashi

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P2860

Hypoxia-inducible factor 1 is a basic-helix-loop-helix-PAS heterodimer regulated by cellular O2 tension

Overexpression of hypoxia-inducible factor 1alpha in common human cancers and their metastases

Identification of genes differentially induced by hypoxia in pancreatic cancer cells

Cancer metastasis: tumor cell and host organ properties important in metastasis to specific secondary sites

Glucose uptake and glycolysis reduce hypoxia-induced apoptosis in cultured neonatal rat cardiac myocytes.

Oncogenic alterations of metabolism.

Expression of hypoxia-inducible factor 1: mechanisms and consequences.

Microenvironment-induced cancer metastasis.

Tumor cell autocrine motility factor is the neuroleukin/phosphohexose isomerase polypeptide.

Tumor cell autocrine motility factor

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