Antitumor mechanisms when pRb and p53 are genetically inactivated. - Wikidata - wikimedia - TriplyDB

Antitumor mechanisms when pRb and p53 are genetically inactivated.

Antitumor mechanisms when pRb and p53 are genetically inactivated.

http://www.wikidata.org/entity/Q38285170

about

Substituting threonine 187 with alanine in p27Kip1 prevents pituitary tumorigenesis by two-hit loss of Rb1 and enhances humoral immunity in old age Bringing Down Cancer Aircraft: Searching for Essential Hypomutated Proteins in Skin Melanoma Aryl hydrocarbon receptor regulates histone deacetylase 8 expression to repress tumor suppressive activity in hepatocellular carcinoma Indirect p53-dependent transcriptional repression of Survivin, CDC25C, and PLK1 genes requires the cyclin-dependent kinase inhibitor p21/CDKN1A and CDE/CHR promoter sites binding the DREAM complex.Thyroid C-Cell Biology and Oncogenic Transformation.Kv10.1 K(+) channel: from physiology to cancer.The p53 tetramer shows an induced-fit interaction of the C-terminal domain with the DNA-binding domain.Effects of concomitant inactivation of p53 and pRb on response to doxorubicin treatment in breast cancer cell lines.Genomic structure, expression, and functional characterization of checkpoint kinase 1 from Penaeus monodon.FGF9/FGFR2 increase cell proliferation by activating ERK1/2, Rb/E2F1, and cell cycle pathways in mouse Leydig tumor cells

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P2860

Antitumor mechanisms when pRb and p53 are genetically inactivated.

http://www.wikidata.org/entity/Q38285170

description

2014 nî lūn-bûn

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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2014年论文

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2014年论文

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name

Antitumor mechanisms when pRb and p53 are genetically inactivated.

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type

label

Antitumor mechanisms when pRb and p53 are genetically inactivated.

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prefLabel

Antitumor mechanisms when pRb and p53 are genetically inactivated.

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Antitumor mechanisms when pRb and p53 are genetically inactivated.

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H Zhao

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L Zhu

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Z Lu

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P2860

Hallmarks of Cancer: The Next Generation

Acetylation-dependent regulation of Skp2 function

E2F7, a novel target, is up-regulated by p53 and mediates DNA damage-dependent transcriptional repression

DNA damage activates a spatially distinct late cytoplasmic cell-cycle checkpoint network controlled by MK2-mediated RNA stabilization

Mutant p53 disrupts mammary tissue architecture via the mevalonate pathway

Phosphate-activated glutaminase (GLS2), a p53-inducible regulator of glutamine metabolism and reactive oxygen species

Glutaminase 2, a novel p53 target gene regulating energy metabolism and antioxidant function

Reciprocal regulation of p53 and malic enzymes modulates metabolism and senescence

Pharmacological inactivation of Skp2 SCF ubiquitin ligase restricts cancer stem cell traits and cancer progression

Proapoptotic function of the retinoblastoma tumor suppressor protein

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10.1038/ONC.2014.399

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35

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34

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2014-12-08T00:00:00Z

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2015-08-27T00:00:00Z

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25486431

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nuclear protein

DNA-binding protein

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neoplasm proteins

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