Alternative splicing of DNA damage response genes and gastrointestinal cancers.
about
Expression of evolutionarily novel genes in tumorsHaploinsufficiency of the c-myc transcriptional repressor FIR, as a dominant negative-alternative splicing model, promoted p53-dependent T-cell acute lymphoblastic leukemia progression by activating Notch1.Anti-FIRs (PUF60) auto-antibodies are detected in the sera of early-stage colon cancer patients.Adenovirus-mediated FIR demonstrated TP53-independent cell-killing effect and enhanced antitumor activity of carbon-ion beams.Disturbed alternative splicing of FIR (PUF60) directed cyclin E overexpression in esophageal cancers.Roles of Smads Family and Alternative Splicing Variants of Smad4 in Different Cancers
P2860
Alternative splicing of DNA damage response genes and gastrointestinal cancers.
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Alternative splicing of DNA damage response genes and gastrointestinal cancers.
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type
label
Alternative splicing of DNA damage response genes and gastrointestinal cancers.
@en
prefLabel
Alternative splicing of DNA damage response genes and gastrointestinal cancers.
@en
P2860
P1476
Alternative splicing of DNA damage response genes and gastrointestinal cancers
@en
P2093
Fumio Nomura
P2860
P304
17305-17313
P356
10.3748/WJG.V20.I46.17305
P407
P577
2014-12-01T00:00:00Z