ABCG1 influences the brain cholesterol biosynthetic pathway but does not affect amyloid precursor protein or apolipoprotein E metabolism in vivo. - Wikidata - wikimedia - TriplyDB

ABCG1 influences the brain cholesterol biosynthetic pathway but does not affect amyloid precursor protein or apolipoprotein E metabolism in vivo.

ABCG1 influences the brain cholesterol biosynthetic pathway but does not affect amyloid precursor protein or apolipoprotein E metabolism in vivo.

http://www.wikidata.org/entity/Q38293052

about

Intracerebral haemorrhage in Down syndrome: protected or predisposed?Apolipoprotein E: from cardiovascular disease to neurodegenerative disorders Dissecting Alzheimer disease in Down syndrome using mouse models A genetic cause of Alzheimer disease: mechanistic insights from Down syndrome Mammalian P4-ATPases and ABC transporters and their role in phospholipid transport Impaired development of atherosclerosis in Abcg1-/- Apoe-/- mice: identification of specific oxysterols that both accumulate in Abcg1-/- Apoe-/- tissues and induce apoptosis.The effects of cholesterol on learning and memory.The vascular contribution to Alzheimer's disease.Apolipoproteins in the brain: implications for neurological and psychiatric disorders.Function and comorbidities of apolipoprotein e in Alzheimer's disease.ABCG1 and ABCG4 Suppress γ-Secretase Activity and Amyloid β Production.APP overexpression in the absence of NPC1 exacerbates metabolism of amyloidogenic proteins of Alzheimer's disease.ABCG1 regulates mouse adipose tissue macrophage cholesterol levels and ratio of M1 to M2 cells in obesity and caloric restriction Dyslipidemia and dementia: current epidemiology, genetic evidence, and mechanisms behind the associations.Reduction of cholesterol synthesis in the mouse brain does not affect amyloid formation in Alzheimer's disease, but does extend lifespan.Cognition, learning behaviour and hippocampal synaptic plasticity are not disrupted in mice over-expressing the cholesterol transporter ABCG1.Emerging new paradigms for ABCG transporters.AMP-activated protein kinase: a potential player in Alzheimer's disease.Molecular basis of etiological implications in Alzheimer's disease: focus on neuroinflammation.Central Nervous System Lipoproteins: ApoE and Regulation of Cholesterol Metabolism.Differential regulation of ABCA1 and ABCG1 gene expressions in the remodeling mouse hippocampus after entorhinal cortex lesion and liver-X receptor agonist treatment The ABCs of sterol transport.Amyloid-β42 alters apolipoprotein E solubility in brains of mice with five familial AD mutations.ABC Transporters Are Key Players in Alzheimer's Disease.

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P2860

ABCG1 influences the brain cholesterol biosynthetic pathway but does not affect amyloid precursor protein or apolipoprotein E metabolism in vivo.

http://www.wikidata.org/entity/Q38293052

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

@zh-sg

2008年學術文章

@yue

2008年學術文章

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2008年學術文章

@zh-hant

name

ABCG1 influences the brain cho ...... oprotein E metabolism in vivo.

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ABCG1 influences the brain cho ...... oprotein E metabolism in vivo.

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ABCG1 influences the brain cho ...... oprotein E metabolism in vivo.

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JLR.M700481-JLR200

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Journal of Lipid Research

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ABCG1 influences the brain cho ...... poprotein E metabolism in vivo

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P2093

Anna Wilkinson

http://www.w3.org/2001/XMLSchema#string

Braydon L Burgess

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Charmaine Wong

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Cheryl L Wellington

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Danielle Balik

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Dieter Lütjohann

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James Donkin

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Jeniffer Y Chan

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Jianjia Fan

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Judes Poirier

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P2860

cDNA cloning of mouse and human cholesterol 25-hydroxylases, polytopic membrane proteins that synthesize a potent oxysterol regulator of lipid metabolism

cDNA cloning of cholesterol 24-hydroxylase, a mediator of cholesterol homeostasis in the brain

Beta-secretase cleavage of Alzheimer's amyloid precursor protein by the transmembrane aspartic protease BACE

Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein

The cholesterol transporter ABCG1 modulates the subcellular distribution and proteolytic processing of beta-amyloid precursor protein

ABCG1 redistributes cell cholesterol to domains removable by high density lipoprotein but not by lipid-depleted apolipoproteins

LXR-induced redistribution of ABCG1 to plasma membrane in macrophages enhances cholesterol mass efflux to HDL

Efflux of sphingomyelin, cholesterol, and phosphatidylcholine by ABCG1

Amyloidogenic processing of the Alzheimer beta-amyloid precursor protein depends on lipid rafts

Gene dose of apolipoprotein E type 4 allele and the risk of Alzheimer's disease in late onset families

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1254-1267

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scholarly article

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10.1194/JLR.M700481-JLR200

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6

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49

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