Modulation of cytokine patterns of human autoreactive T cell clones by a single amino acid substitution of their peptide ligand.
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Posttraumatic therapeutic vaccination with modified myelin self-antigen prevents complete paralysis while avoiding autoimmune diseaseA critical role for transforming growth factor-beta but not interleukin 4 in the suppression of T helper type 1-mediated colitis by CD45RB(low) CD4+ T cellsGlatiramer acetate (Copaxone) induces degenerate, Th2-polarized immune responses in patients with multiple sclerosis.Peptide-based molecular analyses of HLA class II-associated susceptibility to autoimmune diseases.Oral tolerance with copolymer 1 for the treatment of multiple sclerosis.MHC-restricted antigen presentation and recognition: constraints on gene, recombinant and peptide vaccines in humans.Cytotoxic T cells from human immunodeficiency virus type 2-infected patients frequently cross-react with different human immunodeficiency virus type 1 clades.Superantigens: mechanisms by which they may induce, exacerbate and control autoimmune diseases.Analysis of immune responses against T- and B-cell epitopes from Plasmodium falciparum liver-stage antigen 1 in rodent malaria models and malaria-exposed human subjects in India.Selective immunointervention in autoimmune diseases: lessons from multiple sclerosis.Induction of dominant transplantation tolerance by an altered peptide ligand of the male antigen Dby.The physiological role of regulatory T cells in the prevention of autoimmunity: the function of the thymus in the generation of the regulatory T cell subset.Efficient help for autoreactive B-cell activation requires CD4+ T-cell recognition of an agonist peptide at the effector stage.A CD4+ T cell antagonist epitope down-regulates activating signaling proteins, up-regulates inhibitory signaling proteins and abrogates HIV-specific T cell function.Degeneracy, as opposed to specificity, in immunotherapy.Modulation of CD4 T cell function by soluble MHC II-peptide chimeras.Multi-modal antigen specific therapy for autoimmunity.Peptide-based immunotherapy of autoimmunity: a path of puzzles, paradoxes and possibilities.Designing of interferon-gamma inducing MHC class-II binders.Specificity, degeneracy, and molecular mimicry in antigen recognition by HLA-Class II restricted T cell receptors: implications for clinical medicine.Autoimmunity to heat shock protein 60 and antigen-specific production of interleukin-10Long-term production of BDNF and NT-3 induced by A91-immunization after spinal cord injury.Systemic immunomodulation of autoimmune disease using MHC-derived recombinant TCR ligandsAntigen-specific therapies for the treatment of multiple sclerosis: a clinical trial update.Antigen-specific therapies in multiple sclerosis: going beyond proteins and peptides.Endogenous altered peptide ligands can affect peripheral T cell responses.Selective activation of Fas/Fas ligand-mediated cytotoxicity by a self peptidePartial signaling by CD8+ T cells in response to antagonist ligands.Peripheral autoantigen induces regulatory T cells that prevent autoimmunity.Altered ligands reveal limited plasticity in the T cell response to a pathogenic epitope.Microbial epitopes act as altered peptide ligands to prevent experimental autoimmune encephalomyelitisAllelic variation of MHC structure alters peptide ligands to induce atypical partial agonistic CD8+ T cell function.The efficiency of CD4 recruitment to ligand-engaged TCR controls the agonist/partial agonist properties of peptide-MHC molecule ligands.A T helper cell 2 (Th2) immune response against non-self antigens modifies the cytokine profile of autoimmune T cells and protects against experimental allergic encephalomyelitis.Presentation of a T cell receptor antagonist peptide by immunoglobulins ablates activation of T cells by a synthetic peptide or proteins requiring endocytic processingKinetics and extent of T cell activation as measured with the calcium signalPersistence of peptide-induced CD4+ T cell anergy in vitroInhibition of intrathymic T cell development by expression of a transgenic antagonist peptide.The role of CD4-Lck in T-cell receptor antagonism: evidence for negative signalingF(c)gammaRI-targeted fusion proteins result in efficient presentation by human monocytes of antigenic and antagonist T cell epitopes
P2860
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P2860
Modulation of cytokine patterns of human autoreactive T cell clones by a single amino acid substitution of their peptide ligand.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
@zh-cn
name
Modulation of cytokine pattern ...... ution of their peptide ligand.
@en
type
label
Modulation of cytokine pattern ...... ution of their peptide ligand.
@en
prefLabel
Modulation of cytokine pattern ...... ution of their peptide ligand.
@en
P2093
P1433
P1476
Modulation of cytokine pattern ...... ution of their peptide ligand.
@en
P2093
P304
P356
10.1016/1074-7613(95)90145-0
P407
P577
1995-04-01T00:00:00Z