Hypoxia triggers a HIF-mediated differentiation of peripheral blood mononuclear cells into osteoclasts. - Wikidata - wikimedia - TriplyDB

Hypoxia triggers a HIF-mediated differentiation of peripheral blood mononuclear cells into osteoclasts.

Hypoxia triggers a HIF-mediated differentiation of peripheral blood mononuclear cells into osteoclasts.

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The multi-differentiation potential of peripheral blood mononuclear cells The role of hypoxia in orthodontic tooth movement A small molecule with anticancer and antimetastatic activities induces rapid mitochondrial-associated necrosis in breast cancer.Hypoxic regulation of osteoclast differentiation and bone resorption activity A pilot study of change in fracture risk in patients with acute respiratory distress syndrome.The Key Role of the Blood Supply to Bone The Potential Use of Pharmacological Agents to Modulate Orthodontic Tooth Movement (OTM).External cervical resorption: an analysis using cone beam and microfocus computed tomography and scanning electron microscopy.Raloxifene modulates regulators of osteoclastogenesis and angiogenesis in an oestrogen deficiency periapical lesion model.Hypoxia regulates the proliferation and osteogenic differentiation of human periodontal ligament cells under cyclic tensile stress via mitogen-activated protein kinase pathways.Hypoxia Inducible Transcription Factors, HIF1A and HIF2A, Increase in Aging Mucosal Tissues.Models of Bone Remodelling and Associated Disorders

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Hypoxia triggers a HIF-mediated differentiation of peripheral blood mononuclear cells into osteoclasts.

http://www.wikidata.org/entity/Q38328462

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2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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2012年學術文章

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Hypoxia triggers a HIF-mediate ...... uclear cells into osteoclasts.

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Hypoxia triggers a HIF-mediate ...... uclear cells into osteoclasts.

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Hypoxia triggers a HIF-mediate ...... uclear cells into osteoclasts.

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J.1601-6343.2011.01530.X

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Orthodontics & Craniofacial Research

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Hypoxia triggers a HIF-mediate ...... uclear cells into osteoclasts.

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B Disch

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G F Currier

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J Thorpe

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M A Ihnat

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P2860

Bone acid phosphatase: tartrate-resistant acid phosphatase as a marker of osteoclast function

OPGL is a key regulator of osteoclastogenesis, lymphocyte development and lymph-node organogenesis

Osteoclast differentiation and activation

Origin of osteoclasts: mature monocytes and macrophages are capable of differentiating into osteoclasts under a suitable microenvironment prepared by bone marrow-derived stromal cells.

The hypoxia-inducible factor alpha pathway couples angiogenesis to osteogenesis during skeletal development.

Hypoxia-inducible factor-1 (HIF-1).

Monocyte activation in angiogenesis and collateral growth in the rabbit hindlimb.

Hypoxia-inducible factor is expressed in giant cell tumour of bone and mediates paracrine effects of hypoxia on monocyte-osteoclast differentiation via induction of VEGF.

Expression of mRNA for osteoprotegerin and receptor activator of nuclear factor kappa beta ligand (RANKL) during root resorption induced by the application of heavy orthodontic forces on rat molars.

Role of HIF signaling on tumorigenesis in response to chronic low-dose arsenic administration.

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10.1111/J.1601-6343.2011.01530.X

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