The octamer-binding protein Oct-2 represses HSV immediate-early genes in cell lines derived from latently infectable sensory neurons.
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ICP0 antagonizes ICP4-dependent silencing of the herpes simplex virus ICP0 geneOct-1 interacts with conserved motifs in the human thyroid transcription factor 1 gene minimal promoterThe octamer-binding proteins Oct-1 and Oct-2 repress the HIV long terminal repeat promoter and its transactivation by TatThe molecular basis of herpes simplex virus latencyThe ability of POU family transcription factors to activate or repress gene expression is dependent on the spacing and context of their specific response elementsThe Oct-2 transcription factor represses tyrosine hydroxylase expression via a heptamer TAATGARAT-like motif in the gene promoterRole of chromatin during herpesvirus infections.Herpes simplex virus type 1 vector-mediated expression of nerve growth factor protects dorsal root ganglion neurons from peroxide toxicityRegulation of DNA virus transcription by cellular POU family transcription factors.Immunohistochemical analysis of primary sensory neurons latently infected with herpes simplex virus type 1.Latent Marek's disease virus can be activated from its chromosomally integrated state in herpesvirus-transformed lymphoma cells.The neuronal host cell factor-binding protein Zhangfei inhibits herpes simplex virus replication.Transcription factors interacting with herpes simplex virus alpha gene promoters in sensory neuronsNegative regulatory elements upstream of a novel exon of the neuronal nicotinic acetylcholine receptor alpha 2 subunit geneTransactivation of the human papilloma virus 16 octamer motif by the octamer binding protein Oct-2 requires both the N and C terminal activation domains.Experimental investigation of herpes simplex virus latencyProteins and cis-acting elements associated with transactivation of the varicella-zoster virus (VZV) immediate-early gene 62 promoter by VZV open reading frame 10 protein.Neurons differentially control expression of a herpes simplex virus type 1 immediate-early promoter in transgenic mice.Analysis of bovine herpesvirus 1 transcripts during a primary infection of trigeminal ganglia of cattle.Long-term promoter activity during herpes simplex virus latencyCorrelation between precolonization of trigeminal ganglia by attenuated strains of pseudorabies virus and resistance to wild-type virus latency.Altered protein binding to the octamer motif appears to be an early event in programmed neuronal cell death.Herpes simplex virus pathogenesis in transgenic mice is altered by the homeodomain protein Hox 1.3.Induction of reactivation of herpes simplex virus in murine sensory ganglia in vivo by cadmium.Chromatin control of herpes simplex virus lytic and latent infection.Impact of human cytomegalovirus latent infection on myeloid progenitor cell gene expression.Activation of the herpes simplex virus immediate-early gene promoters by neuronally expressed POU family transcription factors.Functional interaction between different isoforms of the Oct-2 transcription factor expressed in neuronal cells.A novel POU family transcription factor is closely related to Brn-3 but has a distinct expression pattern in neuronal cells.The different inhibitory domains of the Oct-2 transcription factor have distinct functional activities.The POU-domain factor Brn-3.0 recognizes characteristic sites in the herpes simplex virus genome.Adjacent proline residues in the inhibitory domain of the Oct-2 transcription factor play distinct functional roles.Role of defective Oct-2 and OCA-B expression in immunoglobulin production and Kaposi's sarcoma-associated herpesvirus lytic reactivation in primary effusion lymphoma.Association of ICP0 but not ICP27 with purified virions of herpes simplex virus type 1.Inhibition of herpes simplex virus infection by ectopic expression of neuronal splice variants of the Oct-2 transcription factor.Functional differences between the Oct2 transactivation domains determine the transactivation potential of individual Oct2 isoforms.The expression of the developmentally regulated proto-oncogene Pax-3 is modulated by N-Myc.Expression of vhs and VP16 during HSV-1 helper virus-free amplicon packaging enhances titers.Human neuron-committed teratocarcinoma NT2 cell line has abnormal ND10 structures and is poorly infected by herpes simplex virus type 1.The DNA binding activity of the paired box transcription factor Pax-3 is rapidly downregulated during neuronal cell differentiation.
P2860
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P2860
The octamer-binding protein Oct-2 represses HSV immediate-early genes in cell lines derived from latently infectable sensory neurons.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年学术文章
@wuu
1991年学术文章
@zh-cn
1991年学术文章
@zh-hans
1991年学术文章
@zh-my
1991年学术文章
@zh-sg
1991年學術文章
@yue
1991年學術文章
@zh
1991年學術文章
@zh-hant
name
The octamer-binding protein Oc ...... ly infectable sensory neurons.
@en
type
label
The octamer-binding protein Oc ...... ly infectable sensory neurons.
@en
prefLabel
The octamer-binding protein Oc ...... ly infectable sensory neurons.
@en
P2093
P1433
P1476
The octamer-binding protein Oc ...... ly infectable sensory neurons.
@en
P2093
Latchman DS
Lillycrop KA
Ninkina NN
Wheatley SC
P304
P356
10.1016/0896-6273(91)90290-G
P407
P577
1991-09-01T00:00:00Z