Neuronal injury mediated via stimulation of microglial toll-like receptor-9 (TLR9).
about
The microglial "activation" continuum: from innate to adaptive responsesEpilepsy and innate immune system: A possible immunogenic predisposition and related therapeutic implicationsMicroglia activated with the toll-like receptor 9 ligand CpG attenuate oligomeric amyloid {beta} neurotoxicity in in vitro and in vivo models of Alzheimer's disease.Expression of a Cu,Zn superoxide dismutase typical for familial amyotrophic lateral sclerosis increases the vulnerability of neuroblastoma cells to infectious injuryToll-like receptor 9 polymorphisms are associated with severity variables in a cohort of meningococcal meningitis survivors.Adrenalectomy promotes a permanent decrease of plasma corticoid levels and a transient increase of apoptosis and the expression of Transforming Growth Factor beta1 (TGF-beta1) in hippocampus: effect of a TGF-beta1 oligo-antisense.Intracellular expression of toll-like receptor 4 in neuroblastoma cells and their unresponsiveness to lipopolysaccharideMicroglial activation mediates host neuronal survival induced by neural stem cells.Plasmacytoid dendritic cells in multiple sclerosis: chemokine and chemokine receptor modulation by interferon-beta.Characterization of a pneumococcal meningitis mouse modelThe role of antigen presenting cells in multiple sclerosis.Toll-like receptors in health and disease in the brain: mechanisms and therapeutic potential.Overview of toll-like receptors in the CNS.Interferon-β inhibits toll-like receptor 9 processing in multiple sclerosis.Toll-like receptor signaling in neural plasticity and disease.Activated microglia proliferate at neurites of mutant huntingtin-expressing neuronsHigher mortality and impaired elimination of bacteria in aged mice after intracerebral infection with E. coli are associated with an age-related decline of microglia and macrophage functions.Extracellular calcium reduction strongly increases the lytic capacity of pneumolysin from streptococcus pneumoniae in brain tissue.Changes in astrocyte shape induced by sublytic concentrations of the cholesterol-dependent cytolysin pneumolysin still require pore-forming capacity.Effects of low dose GM-CSF on microglial inflammatory profiles to diverse pathogen-associated molecular patterns (PAMPs).IFN-gamma signaling in the central nervous system controls the course of experimental autoimmune encephalomyelitis independently of the localization and composition of inflammatory foci.Phase 1 trial of a CpG oligodeoxynucleotide for patients with recurrent glioblastomaTLR9 ligand CpG-ODN applied to the injured mouse cornea elicits retinal inflammation.Emerging role of Toll-like receptors in the control of pain and itch.The biphasic role of microglia in Alzheimer's disease.Toll-like receptors in central nervous system glial inflammation and homeostasis.Plasmacytoid dendritic cells and immunotherapy in multiple sclerosis.Interactions between TLR7 and TLR9 agonists and receptors regulate innate immune responses by astrocytes and microglia.Innate Immunity Stimulation via Toll-Like Receptor 9 Ameliorates Vascular Amyloid Pathology in Tg-SwDI Mice with Associated Cognitive Benefits.Endogenous toll-like receptor ligands and their biological significance.Strategies to increase the activity of microglia as efficient protectors of the brain against infections.Magnesium therapy improves outcome in Streptococcus pneumoniae meningitis by altering pneumolysin pore formation.Removal of pattern-breaking sequences in microtubule binding repeats produces instantaneous tau aggregation and toxicity.Toll-like receptor stimulation increases phagocytosis of Cryptococcus neoformans by microglial cellsToll-like receptors-2 and -9 (TLR2 and TLR9) gene polymorphism in patients with type 2 diabetes and diabetic footIntraperitoneal prophylaxis with CpG oligodeoxynucleotides protects neutropenic mice against intracerebral Escherichia coli K1 infection.Toll-like receptor stimulation enhances phagocytosis and intracellular killing of nonencapsulated and encapsulated Streptococcus pneumoniae by murine microglia.Clearance of apoptotic neurons without inflammation by microglial triggering receptor expressed on myeloid cells-2.Suppressed microglial E prostanoid receptor 1 signaling selectively reduces tumor necrosis factor alpha and interleukin 6 secretion from toll-like receptor 3 activation.Pharmacological Tools to Activate Microglia and their Possible use to Study Neural Network Patho-physiology.
P2860
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P2860
Neuronal injury mediated via stimulation of microglial toll-like receptor-9 (TLR9).
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Neuronal injury mediated via stimulation of microglial toll-like receptor-9 (TLR9).
@en
type
label
Neuronal injury mediated via stimulation of microglial toll-like receptor-9 (TLR9).
@en
prefLabel
Neuronal injury mediated via stimulation of microglial toll-like receptor-9 (TLR9).
@en
P2093
P356
P1433
P1476
Neuronal injury mediated via stimulation of microglial toll-like receptor-9 (TLR9)
@en
P2093
Argyrios K Stringaris
Asparouh I Iliev
Roland Nau
P304
P356
10.1096/FJ.03-0670FJE
P407
P577
2003-12-19T00:00:00Z